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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)

D.J. Grainger

Department of Medicine

Box 157

Addenbrooke's Hospital

Hills Road



Name/email consistency: high



  • Department of Medicine, Box 157, Addenbrooke's Hospital, Hills Road, UK. 2000 - 2002
  • Department of Biochemistry, University of Cambridge, Tennis Court Road, Cambridge, UK. 1998 - 2000


  1. High titres of serum antinuclear antibodies, mostly directed against nucleolar antigens, are associated with the presence of coronary atherosclerosis. Grainger, D.J., Bethell, H.W. Ann. Rheum. Dis. (2002) [Pubmed]
  2. Suppressing thrombin generation is compatible with the development of atherosclerosis in mice. Grainger, D.J., McWilliam, N.A., Baglin, T.P., Byrne, C.D. Thromb. Res. (2001) [Pubmed]
  3. TGF-beta in blood: a complex problem. Grainger, D.J., Mosedale, D.E., Metcalfe, J.C. Cytokine Growth Factor Rev. (2000) [Pubmed]
  4. Dietary fat and reduced levels of TGFbeta1 act synergistically to promote activation of the vascular endothelium and formation of lipid lesions. Grainger, D.J., Mosedale, D.E., Metcalfe, J.C., Böttinger, E.P. J. Cell. Sci. (2000) [Pubmed]
  5. Thrombospondin 1 does not activate transforming growth factor beta1 in a chemically defined system or in smooth-muscle-cell cultures. Grainger, D.J., Frow, E.K. Biochem. J. (2000) [Pubmed]
  6. Transforming growth factor-beta dynamically regulates vascular smooth muscle differentiation in vivo. Grainger, D.J., Metcalfe, J.C., Grace, A.A., Mosedale, D.E. J. Cell. Sci. (1998) [Pubmed]
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