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Natalia I. Dmitrieva

Laboratory of Kidney and Electrolyte Metabolism

National Heart

Lung, and Blood Institute

National Institutes of Health

USA

[email]@nhlbi.nih.gov

Name/email consistency: high

 
 
 
 
 
 
 

Affiliations

  • Laboratory of Kidney and Electrolyte Metabolism, National Heart, Lung, and Blood Institute, National Institutes of Health, USA. 2001 - 2009
  • NHLBI, National Institutes of Health, Bethesda, Maryland 20892-1603 and the Whitney Laboratory, USA. 2000

References

  1. Knockout of Ku86 accelerates cellular senescence induced by high NaCl. Dmitrieva, N.I., Chen, H.T., Nussenzweig, A., Burg, M.B. Aging. (Albany. NY) (2009) [Pubmed]
  2. Analysis of DNA breaks, DNA damage response, and apoptosis produced by high NaCl. Dmitrieva, N.I., Burg, M.B. Am. J. Physiol. Renal Physiol. (2008) [Pubmed]
  3. High NaCl promotes cellular senescence. Dmitrieva, N.I., Burg, M.B. Cell. Cycle (2007) [Pubmed]
  4. The saltiness of the sea breaks DNA in marine invertebrates: possible implications for animal evolution. Dmitrieva, N.I., Ferraris, J.D., Norenburg, J.L., Burg, M.B. Cell. Cycle (2006) [Pubmed]
  5. Ku86 preserves chromatin integrity in cells adapted to high NaCl. Dmitrieva, N.I., Celeste, A., Nussenzweig, A., Burg, M.B. Proc. Natl. Acad. Sci. U.S.A. (2005) [Pubmed]
  6. Cells adapted to high NaCl have many DNA breaks and impaired DNA repair both in cell culture and in vivo. Dmitrieva, N.I., Cai, Q., Burg, M.B. Proc. Natl. Acad. Sci. U.S.A. (2004) [Pubmed]
  7. Living with DNA breaks is an everyday reality for cells adapted to high NaCl. Dmitrieva, N.I., Burg, M.B. Cell. Cycle (2004) [Pubmed]
  8. High NaCl causes Mre11 to leave the nucleus, disrupting DNA damage signaling and repair. Dmitrieva, N.I., Bulavin, D.V., Burg, M.B. Am. J. Physiol. Renal Physiol. (2003) [Pubmed]
  9. Rapid activation of G2/M checkpoint after hypertonic stress in renal inner medullary epithelial (IME) cells is protective and requires p38 kinase. Dmitrieva, N.I., Bulavin, D.V., Fornace, A.J., Burg, M.B. Proc. Natl. Acad. Sci. U.S.A. (2002) [Pubmed]
  10. p53 Protects renal inner medullary cells from hypertonic stress by restricting DNA replication. Dmitrieva, N., Michea, L., Burg, M. Am. J. Physiol. Renal Physiol. (2001) [Pubmed]
  11. Protection of renal inner medullary epithelial cells from apoptosis by hypertonic stress-induced p53 activation. Dmitrieva, N., Kultz, D., Michea, L., Ferraris, J., Burg, M. J. Biol. Chem. (2000) [Pubmed]
 
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