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Yanjun Liu

Division of Endocrinology

Metabolism and Molecular Medicine

UCLA School of Medicine

The Charles Drew University of Medicine and Science

USA

[email]@hotmail.com

Name/email consistency: high

 
 
 
 
 
 
 

Affiliations

  • Division of Endocrinology, Metabolism and Molecular Medicine, UCLA School of Medicine, The Charles Drew University of Medicine and Science, USA. 2008
  • Division of Endocrinology, Charles R. Drew University of Medicine & Sciences, UCLA School of Medicine, 1731 E. 120th Street, USA. 2003 - 2006

References

  1. Reduction of hepatic glucocorticoid receptor and hexose-6-phosphate dehydrogenase expression ameliorates diet-induced obesity and insulin resistance in mice. Liu, Y., Nakagawa, Y., Wang, Y., Liu, L., Du, H., Wang, W., Ren, X., Lutfy, K., Friedman, T.C. J. Mol. Endocrinol. (2008) [Pubmed]
  2. Liver X receptor agonist T0901317 inhibition of glucocorticoid receptor expression in hepatocytes may contribute to the amelioration of diabetic syndrome in db/db mice. Liu, Y., Yan, C., Wang, Y., Nakagawa, Y., Nerio, N., Anghel, A., Lutfy, K., Friedman, T.C. Endocrinology (2006) [Pubmed]
  3. Increased glucocorticoid receptor and 11{beta}-hydroxysteroid dehydrogenase type 1 expression in hepatocytes may contribute to the phenotype of type 2 diabetes in db/db mice. Liu, Y., Nakagawa, Y., Wang, Y., Sakurai, R., Tripathi, P.V., Lutfy, K., Friedman, T.C. Diabetes (2005) [Pubmed]
  4. Leptin activation of corticosterone production in hepatocytes may contribute to the reversal of obesity and hyperglycemia in leptin-deficient ob/ob mice. Liu, Y., Nakagawa, Y., Wang, Y., Li, R., Li, X., Ohzeki, T., Friedman, T.C. Diabetes (2003) [Pubmed]
 
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