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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)
 

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Richard A. Zager

Fred Hutchinson Cancer Research Center

1100 Fairview Ave N; Rm. D2-190

Seattle

WA 98109

[email]@fhcrc.org

Name/email consistency: high

 
 
 
 
 
 
 

Affiliations

  • Fred Hutchinson Cancer Research Center, 1100 Fairview Ave N; Rm. D2-190, Seattle, WA 98109. 2009 - 2012
  • Fred Hutchinson Cancer Research Center and Department of Medicine, University of Washington, Seattle, Washington 98109, USA. 2010
  • Department of Medicine, University of Washington, Seattle, Washington, USA. 1998 - 2010
  • Department of Medicine, University of Washington, and the Clinical Division, Fred Hutchinson Cancer Research Center, USA. 2001 - 2009
  • Department of Medicine, Seattle, USA. 2008

References

  1. Renal cortical hemopexin accumulation in response to acute kidney injury. Zager, R.A., Johnson, A.C., Becker, K. Am. J. Physiol. Renal Physiol. (2012) [Pubmed]
  2. Proximal tubule haptoglobin gene activation is an integral component of the acute kidney injury "stress response". Zager, R.A., Vijayan, A., Johnson, A.C. Am. J. Physiol. Renal Physiol. (2012) [Pubmed]
  3. Plasma and Urinary Heme Oxygenase-1 in AKI. Zager, R.A., Johnson, A.C., Becker, K. J. Am. Soc. Nephrol. (2012) [Pubmed]
  4. Acute unilateral ischemic renal injury induces progressive renal inflammation, lipid accumulation, histone modification, and "end-stage" kidney disease. Zager, R.A., Johnson, A.C., Becker, K. Am. J. Physiol. Renal Physiol. (2011) [Pubmed]
  5. HMG-CoA Reductase Activation and Urinary Pellet Cholesterol Elevations in Acute Kidney Injury. Johnson, A.C., Ware, L.B., Himmelfarb, J., Zager, R.A. Clin. J. Am. Soc. Nephrol (2011) [Pubmed]
  6. Progressive histone alterations and proinflammatory gene activation: consequences of heme protein/iron-mediated proximal tubule injury. Zager, R.A., Johnson, A.C. Am. J. Physiol. Renal Physiol. (2010) [Pubmed]
  7. Acquired cytoresistance in the setting of hematopoietic cell transplantation. Zager, R.A. Clin. J. Am. Soc. Nephrol (2010) [Pubmed]
  8. Uremia impacts renal inflammatory cytokine gene expression in the setting of experimental acute kidney injury. Zager, R.A., Johnson, A.C., Lund, S. Am. J. Physiol. Renal Physiol. (2009) [Pubmed]
  9. Uremia induces proximal tubular cytoresistance and heme oxygenase-1 expression in the absence of acute kidney injury. Zager, R.A. Am. J. Physiol. Renal Physiol. (2009) [Pubmed]
  10. Renal ischemia-reperfusion injury upregulates histone-modifying enzyme systems and alters histone expression at proinflammatory/profibrotic genes. Zager, R.A., Johnson, A.C. Am. J. Physiol. Renal Physiol. (2009) [Pubmed]
  11. Growth and development alter susceptibility to acute renal injury. Zager, R.A., Johnson, A.C., Naito, M., Lund, S.R., Kim, N., Bomsztyk, K. Kidney Int. (2008) [Pubmed]
  12. Maleate nephrotoxicity: mechanisms of injury and correlates with ischemic/hypoxic tubular cell death. Zager, R.A., Johnson, A.C., Naito, M., Bomsztyk, K. Am. J. Physiol. Renal Physiol. (2008) [Pubmed]
  13. 'Endotoxin tolerance': TNF-alpha hyper-reactivity and tubular cytoresistance in a renal cholesterol loading state. Zager, R.A., Johnson, A.C., Lund, S. Kidney Int. (2007) [Pubmed]
  14. "Subclinical" gentamicin nephrotoxicity: a potential risk factor for exaggerated endotoxin-driven TNF-alpha production. Zager, R.A. Am. J. Physiol. Renal Physiol. (2007) [Pubmed]
  15. Toll-like receptor (TLR4) shedding and depletion: acute proximal tubular cell responses to hypoxic and toxic injury. Zager, R.A., Johnson, A.C., Lund, S., Randolph-Habecker, J. Am. J. Physiol. Renal Physiol. (2007) [Pubmed]
  16. Parenteral iron compounds: potent oxidants but mainstays of anemia management in chronic renal disease. Zager, R.A. Clin. J. Am. Soc. Nephrol (2006) [Pubmed]
  17. Acute nephrotoxic and obstructive injury primes the kidney to endotoxin-driven cytokine/chemokine production. Zager, R.A., Johnson, A.C., Hanson, S.Y., Lund, S. Kidney Int. (2006) [Pubmed]
  18. Levosimendan protects against experimental endotoxemic acute renal failure. Zager, R.A., Johnson, A.C., Lund, S., Hanson, S.Y., Abrass, C.K. Am. J. Physiol. Renal Physiol. (2006) [Pubmed]
  19. Acute renal failure: determinants and characteristics of the injury-induced hyperinflammatory response. Zager, R.A., Johnson, A.C., Lund, S., Hanson, S. Am. J. Physiol. Renal Physiol. (2006) [Pubmed]
  20. Parenteral iron compounds sensitize mice to injury-initiated TNF-alpha mRNA production and TNF-alpha release. Zager, R.A., Johnson, A.C., Hanson, S.Y., Lund, S. Am. J. Physiol. Renal Physiol. (2005) [Pubmed]
  21. Renal tubular triglyercide accumulation following endotoxic, toxic, and ischemic injury. Zager, R.A., Johnson, A.C., Hanson, S.Y. Kidney Int. (2005) [Pubmed]
  22. Ischemic proximal tubular injury primes mice to endotoxin-induced TNF-alpha generation and systemic release. Zager, R.A., Johnson, A.C., Hanson, S.Y., Lund, S. Am. J. Physiol. Renal Physiol. (2005) [Pubmed]
  23. Parenteral iron treatment induces MCP-1 accumulation in plasma, normal kidneys, and in experimental nephropathy. Zager, R.A. Kidney Int. (2005) [Pubmed]
  24. Parenteral iron therapy exacerbates experimental sepsis. Zager, R.A., Johnson, A.C., Hanson, S.Y. Kidney Int. (2004) [Pubmed]
  25. Proximal tubular cytochrome c efflux: determinant, and potential marker, of mitochondrial injury. Zager, R.A., Johnson, A.C., Hanson, S.Y. Kidney Int. (2004) [Pubmed]
  26. Parenteral iron nephrotoxicity: potential mechanisms and consequences. Zager, R.A., Johnson, A.C., Hanson, S.Y. Kidney Int. (2004) [Pubmed]
  27. Sepsis syndrome stimulates proximal tubule cholesterol synthesis and suppresses the SR-B1 cholesterol transporter. Zager, R.A., Johnson, A.C., Hanson, S.Y. Kidney Int. (2003) [Pubmed]
  28. Radiographic contrast media-induced tubular injury: evaluation of oxidant stress and plasma membrane integrity. Zager, R.A., Johnson, A.C., Hanson, S.Y. Kidney Int. (2003) [Pubmed]
  29. Acute tubular injury causes dysregulation of cellular cholesterol transport proteins. Zager, R.A., Johnson, A.C., Hanson, S.Y., Shah, V.O. Am. J. Pathol. (2003) [Pubmed]
  30. Proximal tubular cholesterol loading after mitochondrial, but not glycolytic, blockade. Zager, R.A., Johnson, A.C., Hanson, S.Y. Am. J. Physiol. Renal Physiol. (2003) [Pubmed]
  31. Parenteral iron formulations: a comparative toxicologic analysis and mechanisms of cell injury. Zager, R.A., Johnson, A.C., Hanson, S.Y., Wasse, H. Am. J. Kidney Dis. (2002) [Pubmed]
  32. The mevalonate pathway during acute tubular injury: selected determinants and consequences. Zager, R.A., Shah, V.O., Shah, H.V., Zager, P.G., Johnson, A.C., Hanson, S. Am. J. Pathol. (2002) [Pubmed]
  33. Cholesterol ester accumulation: an immediate consequence of acute in vivo ischemic renal injury. Zager, R.A., Johnson, A., Anderson, K., Wright, S. Kidney Int. (2001) [Pubmed]
  34. Renal cholesterol accumulation: a durable response after acute and subacute renal insults. Zager, R.A., Andoh, T., Bennett, W.M. Am. J. Pathol. (2001) [Pubmed]
  35. P glycoprotein-mediated cholesterol cycling determines proximal tubular cell viability. Zager, R.A. Kidney Int. (2001) [Pubmed]
  36. Renal cortical cholesterol accumulation is an integral component of the systemic stress response. Zager, R.A., Johnson, A. Kidney Int. (2001) [Pubmed]
  37. Sphingomyelinase and membrane sphingomyelin content: determinants ofProximal tubule cell susceptibility to injury. Zager, R.A., Burkhart, K.M., Johnson, A. J. Am. Soc. Nephrol. (2000) [Pubmed]
  38. Plasma membrane cholesterol: a critical determinant of cellular energetics and tubular resistance to attack. Zager, R.A. Kidney Int. (2000) [Pubmed]
  39. Polyene antibiotics: relative degrees of in vitro cytotoxicity and potential effects on tubule phospholipid and ceramide content. Zager, R.A. Am. J. Kidney Dis. (2000) [Pubmed]
  40. Changes in free and esterified cholesterol: hallmarks of acute renal tubular injury and acquired cytoresistance. Zager, R.A., Kalhorn, T.F. Am. J. Pathol. (2000) [Pubmed]
  41. Calcitriol directly sensitizes renal tubular cells to ATP-depletion- and iron-mediated attack. Zager, R.A. Am. J. Pathol. (1999) [Pubmed]
  42. Plasma membrane phospholipid integrity and orientation during hypoxic and toxic proximal tubular attack. Zager, R.A., Sacks, B.M., Burkhart, K.M., Williams, A.C. Kidney Int. (1999) [Pubmed]
  43. Increased proximal tubular cholesterol content: implications for cell injury and "acquired cytoresistance". Zager, R.A., Burkhart, K.M., Johnson, A.C., Sacks, B.M. Kidney Int. (1999) [Pubmed]
  44. Isoflurane alters proximal tubular cell susceptibility to toxic and hypoxic forms of attack. Zager, R.A., Burkhart, K.M., Conrad, D.S. Kidney Int. (1999) [Pubmed]
  45. Altered sphingomyelinase and ceramide expression in the setting of ischemic and nephrotoxic acute renal failure. Zager, R.A., Conrad, S., Lochhead, K., Sweeney, E.A., Igarashi, Y., Burkhart, K.M. Kidney Int. (1998) [Pubmed]
  46. Differential effects of glutathione and cysteine on Fe2+, Fe3+, H2O2 and myoglobin-induced proximal tubular cell attack. Zager, R.A., Burkhart, K.M. Kidney Int. (1998) [Pubmed]
 
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