Matthew J. Eagleton
Deparment of Vascular Surgery
Cleveland Clinic Foundation
Cleveland
Ohio 44195
USA
Name/email consistency: high
- Loss of STAT1 is associated with increased aortic rupture in an experimental model of aortic dissection and aneurysm formation. Eagleton, M.J., Xu, J., Liao, M., Parine, B., Chisolm, G.M., Graham, L.M. J. Vasc. Surg. (2010)
- Spinal and visceral ischemia after endovascular aortic repair. Eagleton, M.J., Greenberg, R.K. J. Cardiovasc. Surg. (Torino) (2010)
- Late complications after endovascular thoracoabdominal aneurysm repair. Eagleton, M.J., Greenberg, R.K. Semin. Vasc. Surg (2009)
- Calcium channel blockers and angiotensin-converting enzyme inhibitors may be associated with altered atherosclerotic plaque size and morphology. Eagleton, M.J., Bishop, P.D., Bena, J.F., Nassoiy, S.P., Clair, D.G., Kashyap, V.S., Ouriel, K. Vascular (2008)
- Molecular diagnoses and treatments--past, present, or future?. Eagleton, M.J. Semin. Vasc. Surg (2007)
- Alterations in angiotensin converting enzyme during rodent aortic aneurysm formation. Eagleton, M.J., Cho, B., Lynch, E., Roelofs, K., Woodrum, D., Stanley, J.C., Upchurch, G.R. J. Surg. Res. (2006)
- Early increased MT1-MMP expression and late MMP-2 and MMP-9 activity during Angiotensin II induced aneurysm formation. Eagleton, M.J., Ballard, N., Lynch, E., Srivastava, S.D., Upchurch, G.R., Stanley, J.C. J. Surg. Res. (2006)