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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)
 

Barbara Ahlemeyer

Institut für Pharmakologie und Toxikologie

Fachbereich Pharmazie der Philipps-Universität Marburg

Germany

[email]@*.uni-marburg.de

Name/email consistency: high

 
 
 
 
 
 
 

Affiliation

  • Institut für Pharmakologie und Toxikologie, Fachbereich Pharmazie der Philipps-Universität Marburg, Germany. 1997 - 2002

References

  1. Release of cytochrome c into the extracellular space contributes to neuronal apoptosis induced by staurosporine. Ahlemeyer, B., Klumpp, S., Krieglstein, J. Brain Res. (2002) [Pubmed]
  2. Increase in glutamate-induced neurotoxicity by activated astrocytes involves stimulation of protein kinase C. Ahlemeyer, B., Kölker, S., Zhu, Y., Hoffmann, G.F., Krieglstein, J. J. Neurochem. (2002) [Pubmed]
  3. Neuroprotective effects of NV-31, a bilobalide-derived compound: evidence for an antioxidative mechanism. Ahlemeyer, B., Junker, V., Hühne, R., Krieglstein, J. Brain Res. (2001) [Pubmed]
  4. Retinoic acid reduces apoptosis and oxidative stress by preservation of SOD protein level. Ahlemeyer, B., Bauerbach, E., Plath, M., Steuber, M., Heers, C., Tegtmeier, F., Krieglstein, J. Free Radic. Biol. Med. (2001) [Pubmed]
  5. Inhibition of glutathione depletion by retinoic acid and tocopherol protects cultured neurons from staurosporine-induced oxidative stress and apoptosis. Ahlemeyer, B., Krieglstein, J. Neurochem. Int. (2000) [Pubmed]
  6. S-100beta protects cultured neurons against glutamate- and staurosporine-induced damage and is involved in the antiapoptotic action of the 5 HT(1A)-receptor agonist, Bay x 3702. Ahlemeyer, B., Beier, H., Semkova, I., Schaper, C., Krieglstein, J. Brain Res. (2000) [Pubmed]
  7. Retinoic acid potentiated the protective effect of NGF against staurosporine-induced apoptosis in cultured chick neurons by increasing the trkA protein expression. Ahlemeyer, B., Hühne, R., Krieglstein, J. J. Neurosci. Res. (2000) [Pubmed]
  8. Staurosporine-induced apoptosis in cultured chick embryonic neurons is reduced by polyethylenimine of low molecular weight used as a coating substrate. Ahlemeyer, B., Fischer, D., Kissel, T., Krieglstein, J. Neurosci. Res. (2000) [Pubmed]
  9. The 5-HT1A receptor agonist Bay x 3702 inhibits apoptosis induced by serum deprivation in cultured neurons. Ahlemeyer, B., Glaser, A., Schaper, C., Semkova, I., Krieglstein, J. Eur. J. Pharmacol. (1999) [Pubmed]
  10. Retinoic acid reduces staurosporine-induced apoptotic damage in chick embryonic neurons by suppressing reactive oxygen species production. Ahlemeyer, B., Krieglstein, J. Neurosci. Lett. (1998) [Pubmed]
  11. Stimulation of 5-HT1A receptor inhibits apoptosis induced by serum deprivation in cultured neurons from chick embryo. Ahlemeyer, B., Krieglstein, J. Brain Res. (1997) [Pubmed]
 
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