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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)
 

Elizabeth Kudlacz

Department of Immunology

Pfizer Global Research and Development

Groton

Connecticut 06340

USA

[email]@*.pfizer.com

Name/email consistency: high

 
 
 
 
 
 
 

Affiliation

  • Department of Immunology, Pfizer Global Research and Development, Groton, Connecticut 06340, USA. 2001 - 2004

References

  1. The novel JAK-3 inhibitor CP-690550 is a potent immunosuppressive agent in various murine models. Kudlacz, E., Perry, B., Sawyer, P., Conklyn, M., McCurdy, S., Brissette, W., Flanagan And, M., Changelian, P. Am. J. Transplant. (2004) [Pubmed]
  2. Pulmonary eosinophilia in a murine model of allergic inflammation is attenuated by small molecule alpha4beta1 antagonists. Kudlacz, E., Whitney, C., Andresen, C., Duplantier, A., Beckius, G., Chupak, L., Klein, A., Kraus, K., Milici, A. J. Pharmacol. Exp. Ther. (2002) [Pubmed]
  3. Functional effects of eotaxin are selectively upregulated on IL-5 transgenic mouse eosinophils. Kudlacz, E., Whitney, C., Andresenl, C., Conklyn, M. Inflammation (2002) [Pubmed]
  4. Genetic ablation of the src kinase p59fynT exacerbates pulmonary inflammation in an allergic mouse model. Kudlacz, E.M., Andresen, C.J., Salafia, M., Whitney, C.A., Naclerio, B., Changelian, P.S. Am. J. Respir. Cell Mol. Biol. (2001) [Pubmed]
 
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