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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)
 

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Evangeline D. Motley

Department of Biomedical Sciences

Division of Cardiovascular Biology

Meharry Medical College

Nashville

USA

[email]@mmc.edu

Name/email consistency: high

 
 
 
 
 
 
 

Affiliations

  • Department of Biomedical Sciences, Division of Cardiovascular Biology, Meharry Medical College, Nashville, USA. 2007
  • Department of Anatomy & Physiology, Meharry Medical College, Nashville, TN 37208, USA. 2001 - 2003

References

  1. Mechanism of endothelial nitric oxide synthase phosphorylation and activation by thrombin. Motley, E.D., Eguchi, K., Patterson, M.M., Palmer, P.D., Suzuki, H., Eguchi, S. Hypertension (2007) [Pubmed]
  2. Insulin-induced Akt activation is inhibited by angiotensin II in the vasculature through protein kinase C-alpha. Motley, E.D., Eguchi, K., Gardner, C., Hicks, A.L., Reynolds, C.M., Frank, G.D., Mifune, M., Ohba, M., Eguchi, S. Hypertension (2003) [Pubmed]
  3. Lysophosphatidylcholine inhibits insulin-induced Akt activation through protein kinase C-alpha in vascular smooth muscle cells. Motley, E.D., Kabir, S.M., Gardner, C.D., Eguchi, K., Frank, G.D., Kuroki, T., Ohba, M., Yamakawa, T., Eguchi, S. Hypertension (2002) [Pubmed]
  4. Signaling mechanisms of heparin-binding epidermal growth factor-like growth factor in vascular smooth muscle cells. Reynolds, C.M., Eguchi, S., Frank, G.D., Motley, E.D. Hypertension (2002) [Pubmed]
  5. Protein kinase C inhibits insulin-induced Akt activation in vascular smooth muscle cells. Motley, E.D., Kabir, S.M., Eguchi, K., Hicks, A.L., Gardner, C.D., Reynolds, C.M., Frank, G.D., Eguchi, S. Cell. Mol. Biol. (Noisy-le-grand) (2001) [Pubmed]
 
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