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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)
 

Edward B. Stephens

Department of Anatomy and Cell Biology

University of Kansas Medical Center

5007 Wahl Hall West

Kansas City

USA

[email]@kumc.edu

Name/email consistency: high

 
 
 
 
 
 
 

Affiliations

  • Department of Anatomy and Cell Biology, University of Kansas Medical Center, 5007 Wahl Hall West, Kansas City, USA. 2010
  • Department of Microbiology, Molecular Genetics, and Immunology, University of Kansas Medical Center, USA. 1997 - 2004

References

  1. The vpu protein: new concepts in virus release and CD4 down-modulation. Ruiz, A., Guatelli, J.C., Stephens, E.B. Curr. HIV Res. (2010) [Pubmed]
  2. Vpu: a multifunctional protein that enhances the pathogenesis of human immunodeficiency virus type 1. Hout, D.R., Mulcahy, E.R., Pacyniak, E., Gomez, L.M., Gomez, M.L., Stephens, E.B. Curr. HIV Res. (2004) [Pubmed]
  3. The primary phase of infection by pathogenic simian-human immunodeficiency virus results in disruption of the blood-brain barrier. Stephens, E.B., Singh, D.K., Kohler, M.E., Jackson, M., Pacyniak, E., Berman, N.E. AIDS Res. Hum. Retroviruses (2003) [Pubmed]
  4. Deletion of the vpu sequences prior to the env in a simian-human immunodeficiency virus results in enhanced Env precursor synthesis but is less pathogenic for pig-tailed macaques. Stephens, E.B., McCormick, C., Pacyniak, E., Griffin, D., Pinson, D.M., Sun, F., Nothnick, W., Wong, S.W., Gunderson, R., Berman, N.E., Singh, D.K. Virology (2002) [Pubmed]
  5. Comparison of Vif sequences from diverse geographical isolates of HIV type 1 and SIV(cpz) identifies substitutions common to subtype C isolates and extensive variation in a proposed nuclear transport inhibition signal. Stephens, E.B., Singh, D.K., Pacyniak, E., McCormick, C. AIDS Res. Hum. Retroviruses (2001) [Pubmed]
  6. Simian-human immunodeficiency virus-associated nephropathy in macaques. Stephens, E.B., Tian, C., Dalton, S.B., Gattone, V.H. AIDS Res. Hum. Retroviruses (2000) [Pubmed]
  7. Simian-human immunodeficiency virus (SHIV) containing the nef/long terminal repeat region of the highly virulent SIVsmmPBj14 causes PBj-like activation of cultured resting peripheral blood mononuclear cells, but the chimera showed No increase in virulence. Stephens, E.B., Mukherjee, S., Liu, Z.Q., Sheffer, D., Lamb-Wharton, R., Leung, K., Zhuge, W., Joag, S.V., Li, Z., Foresman, L., Adany, I., Narayan, O. J. Virol. (1998) [Pubmed]
  8. Nucleotide substitutions in the long terminal repeat are not required for development of neurovirulence by simian immunodeficiency virus strain mac. Stephens, E.B., Sahni, M., Leung, K., Raghavan, R., Joag, S.V., Narayan, O. J. Gen. Virol. (1998) [Pubmed]
  9. Rhesus macaques infected with macrophage-tropic simian immunodeficiency virus (SIVmacR71/17E) exhibit extensive focal segmental and global glomerulosclerosis. Stephens, E.B., Tian, C., Li, Z., Narayan, O., Gattone, V.H. J. Virol. (1998) [Pubmed]
  10. Infected macaques that controlled replication of SIVmac or nonpathogenic SHIV developed sterilizing resistance against pathogenic SHIV(KU-1). Stephens, E.B., Joag, S.V., Atkinson, B., Sahni, M., Li, Z., Foresman, L., Adany, I., Narayan, O. Virology (1997) [Pubmed]
 
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