Frances M. Ashcroft
University Laboratory of Physiology
Oxford OX1 3PT
UK
Name/email consistency: high
- K(ATP) channels and insulin secretion: a key role in health and disease. Ashcroft, F.M. Biochem. Soc. Trans. (2006)
- From molecule to malady. Ashcroft, F.M. Nature (2006)
- Functional analysis of six Kir6.2 (KCNJ11) mutations causing neonatal diabetes. Girard, C.A., Shimomura, K., Proks, P., Absalom, N., Castano, L., Perez de Nanclares, G., Ashcroft, F.M. Pflugers Arch. (2006)
- Keeping the heart going: a new role for KATP channels. Tammaro, P., Ashcroft, F.M. J. Physiol. (Lond.) (2006)
- ATP-sensitive potassium channelopathies: focus on insulin secretion. Ashcroft, F.M. J. Clin. Invest. (2005)
- Activating mutations in Kir6.2 and neonatal diabetes: new clinical syndromes, new scientific insights, and new therapy. Hattersley, A.T., Ashcroft, F.M. Diabetes (2005)
- Type 2 diabetes mellitus: not quite exciting enough?. Ashcroft, F., Rorsman, P. Hum. Mol. Genet. (2004)
- Tissue-specific effects of sulfonylureas: lessons from studies of cloned K(ATP) channels. Ashcroft, F.M., Gribble, F.M. J. Diabetes Complicat. (2000)
- New windows on the mechanism of action of K(ATP) channel openers. Ashcroft, F.M., Gribble, F.M. Trends Pharmacol. Sci. (2000)
- Exciting times for PIP2. Ashcroft, F.M. Science (1998)