Giti Garthwaite
Wolfson Institute for Biomedical Research
University College London
UK
Name/email consistency: high
- Signaling from blood vessels to CNS axons through nitric oxide. Garthwaite, G., Bartus, K., Malcolm, D., Goodwin, D., Kollb-Sielecka, M., Kollb-Sielecka, M., Dooldeniya, C., Garthwaite, J. J. Neurosci. (2006)
- Pathological implications of iNOS expression in central white matter: an ex vivo study of optic nerves from rats with experimental allergic encephalomyelitis. Garthwaite, G., Batchelor, A.M., Goodwin, D.A., Hewson, A.K., Leeming, K., Ahmed, Z., Cuzner, M.L., Garthwaite, J. Eur. J. Neurosci. (2005)
- Soluble guanylyl cyclase activator YC-1 protects white matter axons from nitric oxide toxicity and metabolic stress, probably through Na(+) channel inhibition. Garthwaite, G., Goodwin, D.A., Neale, S., Riddall, D., Garthwaite, J. Mol. Pharmacol. (2002)
- Nitric oxide toxicity in CNS white matter: an in vitro study using rat optic nerve. Garthwaite, G., Goodwin, D.A., Batchelor, A.M., Leeming, K., Garthwaite, J. Neuroscience (2002)
- Mechanisms of ischaemic damage to central white matter axons: a quantitative histological analysis using rat optic nerve. Garthwaite, G., Brown, G., Batchelor, A.M., Goodwin, D.A., Garthwaite, J. Neuroscience (1999)
