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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)
 

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G.A. Homandberg

Department of Biochemistry

Rush Medical College

Rush-Presbyterian-St. Luke's Medical Center

Chicago

USA

[email]@rush.edu

Name/email consistency: high

 
 
 
 
 
 
 

Affiliation

  • Department of Biochemistry, Rush Medical College, Rush-Presbyterian-St. Luke's Medical Center, Chicago, USA. 1997 - 2002

References

  1. Antisense oligonucleotides to the integrin receptor subunit alpha(5) decrease fibronectin fragment mediated cartilage chondrolysis. Homandberg, G.A., Costa, V., Ummadi, V., Pichika, R. Osteoarthr. Cartil. (2002) [Pubmed]
  2. Potential regulation of cartilage metabolism in osteoarthritis by fibronectin fragments. Homandberg, G.A. Front. Biosci. (1999) [Pubmed]
  3. Exposure of cartilage to a fibronectin fragment amplifies catabolic processes while also enhancing anabolic processes to limit damage. Homandberg, G.A., Wen, C. J. Orthop. Res. (1998) [Pubmed]
  4. Agents that block fibronectin fragment-mediated cartilage damage also promote repair. Homandberg, G.A., Wen, C., Hui, F. Inflamm. Res. (1997) [Pubmed]
 
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