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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)
 

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Gregg D. Stanwood

Department of Pharmacology

Vanderbilt Kennedy Center

23rd Ave South

476 RRB

USA

[email]@vanderbilt.edu

Name/email consistency: high

 
 
 
 
 
 
 

Affiliations

  • Department of Pharmacology, Vanderbilt Kennedy Center, 23rd Ave South, 476 RRB, USA. 2008
  • John F. Kennedy Center for Research on Human Development and Department of Pharmacology, Vanderbilt University School of Medicine, 8110 Medical Research Building III, Nashville, USA. 2003 - 2007
  • Vanderbilt Kennedy Center for Research on Human Development, Vanderbilt University, Nashville, Tennessee 37203, USA. 2005 - 2007

References

  1. Protein-protein interactions and dopamine D2 receptor signaling: a calcium connection. Stanwood, G.D. Mol. Pharmacol. (2008) [Pubmed]
  2. Prenatal exposure to cocaine produces unique developmental and long-term adaptive changes in dopamine D1 receptor activity and subcellular distribution. Stanwood, G.D., Levitt, P. J. Neurosci. (2007) [Pubmed]
  3. Waved-1 mutant mice are hypersensitive to the locomotor actions of cocaine. Stanwood, G.D., Levitt, P. Synapse (2007) [Pubmed]
  4. Genetic or pharmacological inactivation of the dopamine D1 receptor differentially alters the expression of regulator of G-protein signalling (Rgs) transcripts. Stanwood, G.D., Parlaman, J.P., Levitt, P. Eur. J. Neurosci. (2006) [Pubmed]
  5. Anatomical abnormalities in dopaminoceptive regions of the cerebral cortex of dopamine D1 receptor mutant mice. Stanwood, G.D., Parlaman, J.P., Levitt, P. J. Comp. Neurol. (2005) [Pubmed]
  6. Drug exposure early in life: functional repercussions of changing neuropharmacology during sensitive periods of brain development. Stanwood, G.D., Levitt, P. Curr. Opin. Pharmacol (2004) [Pubmed]
  7. Repeated i.v. cocaine exposure produces long-lasting behavioral sensitization in pregnant adults, but behavioral tolerance in their offspring. Stanwood, G.D., Levitt, P. Neuroscience (2003) [Pubmed]
 
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