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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)

Heather M. Wilson

1.University of Aberdeen

Institute of Medical Sciences





Name/email consistency: high



  • 1.University of Aberdeen, Institute of Medical Sciences, Foresterhill, Aberdeen, UK. 2010 - 2011
  • Section of Immunology and Infection, School of Medicine and Dentistry, Division of Applied Medicine, University of Aberdeen, UK. 2009
  • Department of Medicine and Therapeutics, University of Aberdeen, Aberdeen, Scotland, United Kingdom. 2000 - 2005
  • Department of Molecular and Cell Biology, University of Aberdeen, UK. 1997


  1. Suppressor of cytokine signaling (SOCS)1 is a key determinant of differential macrophage activation and function. Whyte, C.S., Bishop, E.T., Rückerl, D., Gaspar-Pereira, S., Barker, R.N., Allen, J.E., Rees, A.J., Wilson, H.M. J. Leukoc. Biol. (2011) [Pubmed]
  2. Macrophages heterogeneity in atherosclerosis - implications for therapy. Wilson, H.M. J. Cell. Mol. Med. (2010) [Pubmed]
  3. Macrophages: promising targets for the treatment of atherosclerosis. Wilson, H.M., Barker, R.N., Erwig, L.P. Curr. Vasc. Pharmacol (2009) [Pubmed]
  4. Inhibition of macrophage nuclear factor-kappaB leads to a dominant anti-inflammatory phenotype that attenuates glomerular inflammation in vivo. Wilson, H.M., Chettibi, S., Jobin, C., Walbaum, D., Rees, A.J., Kluth, D.C. Am. J. Pathol. (2005) [Pubmed]
  5. Macrophages and the kidney. Wilson, H.M., Walbaum, D., Rees, A.J. Curr. Opin. Nephrol. Hypertens. (2004) [Pubmed]
  6. Bone-marrow-derived macrophages genetically modified to produce IL-10 reduce injury in experimental glomerulonephritis. Wilson, H.M., Stewart, K.N., Brown, P.A., Anegon, I., Chettibi, S., Rees, A.J., Kluth, D.C. Mol. Ther. (2002) [Pubmed]
  7. Transforming growth factor-beta isoforms and glomerular injury in nephrotoxic nephritis. Wilson, H.M., Minto, A.W., Brown, P.A., Erwig, L.P., Rees, A.J. Kidney Int. (2000) [Pubmed]
  8. Opposing effects of interleukin-1 and transforming growth factor-beta on the regulation of tissue-type plasminogen activator and plasminogen activator inhibitor type-1 expression by human mesangial cells. Wilson, H.M., Haites, N.E., Booth, N.A. Exp. Nephrol. (1997) [Pubmed]
  9. Effect of angiotensin II on plasminogen activator inhibitor-1 production by cultured human mesangial cells. Wilson, H.M., Haites, N.E., Booth, N.A. Nephron (1997) [Pubmed]
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