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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)

Haiyan Wang

Division of Clinical Biochemistry

Department of Internal Medicine

University Medical Center

CH-1211 Geneva 4



Name/email consistency: high



  • Division of Clinical Biochemistry, Department of Internal Medicine, University Medical Center, CH-1211 Geneva 4, Switzerland. 2002 - 2005
  • Department of Cell Physiology and Metabolism, University Medical Center, 1211 Geneva 4, Switzerland. 2005


  1. Suppression of Pdx-1 perturbs proinsulin processing, insulin secretion and GLP-1 signalling in INS-1 cells. Wang, H., Iezzi, M., Theander, S., Antinozzi, P.A., Gauthier, B.R., Halban, P.A., Wollheim, C.B. Diabetologia (2005) [Pubmed]
  2. Does chasing selected 'Fox' to the nucleus prevent diabetes?. Wang, H., Wollheim, C.B. Trends. Mol. Med (2005) [Pubmed]
  3. The transcription factor SREBP-1c is instrumental in the development of beta-cell dysfunction. Wang, H., Maechler, P., Antinozzi, P.A., Herrero, L., Hagenfeldt-Johansson, K.A., Bjorklund, A., Wollheim, C.B. J. Biol. Chem. (2003) [Pubmed]
  4. Foxa2 (HNF3beta ) controls multiple genes implicated in metabolism-secretion coupling of glucose-induced insulin release. Wang, H., Gauthier, B.R., Hagenfeldt-Johansson, K.A., Iezzi, M., Wollheim, C.B. J. Biol. Chem. (2002) [Pubmed]
  5. ChREBP rather than USF2 regulates glucose stimulation of endogenous L-pyruvate kinase expression in insulin-secreting cells. Wang, H., Wollheim, C.B. J. Biol. Chem. (2002) [Pubmed]
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