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Masakazu Haneda

Third Department of Medicine

Shiga University of Medical Science

Japan

[email]@*.shiga-med.ac.jp

Name/email consistency: high

 
 
 
 
 
 
 

Affiliation

  • Third Department of Medicine, Shiga University of Medical Science, Japan. 1997 - 2003

References

  1. Overview of glucose signaling in mesangial cells in diabetic nephropathy. Haneda, M., Koya, D., Isono, M., Kikkawa, R. J. Am. Soc. Nephrol. (2003) [Pubmed]
  2. Cellular mechanisms in the development and progression of diabetic nephropathy: activation of the DAG-PKC-ERK pathway. Haneda, M., Koya, D., Kikkawa, R. Am. J. Kidney Dis. (2001) [Pubmed]
  3. Mesangial cell dysfunction as a pathogenesis of diabetic nephropathy. Haneda, M., Koya, D., Kikkawa, R. Contrib. Nephrol (2001) [Pubmed]
  4. Mitogen-activated protein kinase phosphatase: a negative regulator of the mitogen-activated protein kinase cascade. Haneda, M., Sugimoto, T., Kikkawa, R. Eur. J. Pharmacol. (1999) [Pubmed]
  5. Mitogen-activated protein kinase cascade is activated in glomeruli of diabetic rats and glomerular mesangial cells cultured under high glucose conditions. Haneda, M., Araki, S., Togawa, M., Sugimoto, T., Isono, M., Kikkawa, R. Diabetes (1997) [Pubmed]
  6. Activation of mitogen-activated protein kinase cascade in diabetic glomeruli and mesangial cells cultured under high glucose conditions. Haneda, M., Araki, S., Togawa, M., Sugimoto, T., Isono, M., Kikkawa, R. Kidney Int. Suppl. (1997) [Pubmed]
 
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