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Grant M. Hatch

Dept. of Pharmacology and Therapeutics

753 McDermot Ave.

Faculty of Medicine

Univ. of Manitoba

[email]@ms.umanitoba.ca

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Affiliations

Dept. of Pharmacology and Therapeutics, 753 McDermot Ave., Faculty of Medicine, Univ. of Manitoba. 2011
Department of Pharmacology and Therapeutics, University of Manitoba, Winnipeg, Canada. 1998 - 2004
Department of Pharmacology and Therapeutics, and Center for Research and Treatment of Atherosclerosis, University of Manitoba, Winnipeg, Canada. 2004

References

Persistent pulmonary hypertension results in reduced tetralinoleoyl-cardiolipin and mitochondrial complex II + III during the development of right ventricular hypertrophy in the neonatal pig heart. Saini-Chohan, H.K., Dakshinamurti, S., Taylor, W.A., Shen, G.X., Murphy, R., Sparagna, G.C., Hatch, G.M. Am. J. Physiol. Heart Circ. Physiol. (2011) [Pubmed]
C. trachomatis-infection accelerates metabolism of phosphatidylcholine derived from low density lipoprotein but does not affect phosphatidylcholine secretion from hepatocytes. Hatch, G.M., McClarty, G. BMC Microbiol. (2004) [Pubmed]
Cell biology of cardiac mitochondrial phospholipids. Hatch, G.M. Biochem. Cell Biol. (2004) [Pubmed]
AGI-1067. AtheroGenics. Hatch, G.M. Curr. Opin. Investig. Drugs (2002) [Pubmed]
Cardiolipin remodeling in eukaryotic cells infected with Chlamydia trachomatis is linked to elevated mitochondrial metabolism. Hatch, G.M., McClarty, G. Biochem. Biophys. Res. Commun. (1998) [Pubmed]