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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)
 

Henrik Fagman

Department of Medical Chemistry and Cell Biology

Institute of Biomedicine

Sahlgrenska Academy at Göteborg University

SE-40530

Sweden

[email]@anatcell.gu.se

Name/email consistency: high

 
 
 
 
 
 
 

Affiliations

  • Department of Medical Chemistry and Cell Biology, Institute of Biomedicine, Sahlgrenska Academy at Göteborg University, SE-40530, Sweden. 2007
  • Institute of Anatomy and Cell Biology, Goteborg University, SE-40530 Goteborg, Sweden. 2003 - 2006

References

  1. The 22q11 deletion syndrome candidate gene Tbx1 determines thyroid size and positioning. Fagman, H., Liao, J., Westerlund, J., Andersson, L., Morrow, B.E., Nilsson, M. Hum. Mol. Genet. (2007) [Pubmed]
  2. The developing mouse thyroid: embryonic vessel contacts and parenchymal growth pattern during specification, budding, migration, and lobulation. Fagman, H., Andersson, L., Nilsson, M. Dev. Dyn. (2006) [Pubmed]
  3. Genetic deletion of sonic hedgehog causes hemiagenesis and ectopic development of the thyroid in mouse. Fagman, H., Grände, M., Gritli-Linde, A., Nilsson, M. Am. J. Pathol. (2004) [Pubmed]
  4. Expression of classical cadherins in thyroid development: maintenance of an epithelial phenotype throughout organogenesis. Fagman, H., Grände, M., Edsbagge, J., Semb, H., Nilsson, M. Endocrinology (2003) [Pubmed]
  5. Nuclear accumulation of full-length and truncated adenomatous polyposis coli protein in tumor cells depends on proliferation. Fagman, H., Larsson, F., Arvidsson, Y., Meuller, J., Nordling, M., Martinsson, T., Helmbrecht, K., Brabant, G., Nilsson, M. Oncogene (2003) [Pubmed]
 
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