Hiroshi Nagai
Department of Internal Related
Kobe University Graduate School of Medicine
Japan
Name/email consistency: high
- Antitumor activities of interleukin-27 on melanoma. Nagai, H., Oniki, S., Fujiwara, S., Xu, M., Mizoguchi, I., Yoshimoto, T., Nishigori, C. Endocr. Metab. Immune. Disord. Drug. Targets (2010)
- Induction of cellular immunity against hair follicle melanocyte causes alopecia. Nagai, H., Oniki, S., Oka, M., Horikawa, T., Nishigori, C. Arch. Dermatol. Res. (2006)
- In vivo elimination of CD25+ regulatory T cells leads to tumor rejection of B16F10 melanoma, when combined with interleukin-12 gene transfer. Nagai, H., Horikawa, T., Hara, I., Fukunaga, A., Oniki, S., Oka, M., Nishigori, C., Ichihashi, M. Exp. Dermatol. (2004)
- Gene transfer of secreted-type modified interleukin-18 gene to B16F10 melanoma cells suppresses in vivo tumor growth through inhibition of tumor vessel formation. Nagai, H., Hara, I., Horikawa, T., Oka, M., Kamidono, S., Ichihashi, M. J. Invest. Dermatol. (2002)
- Elimination of CD4(+) T cells enhances anti-tumor effect of locally secreted interleukin-12 on B16 mouse melanoma and induces vitiligo-like coat color alteration. Nagai, H., Hara, I., Horikawa, T., Oka, M., Kamidono, S., Ichihashi, M. J. Invest. Dermatol. (2000)