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Hirotaka Osada

Division of Molecular Oncology

Aichi Cancer Center Research Institute

Nagoya 464-8681

Japan

[email]@aichi-cc.jp

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Affiliation

Division of Molecular Oncology, Aichi Cancer Center Research Institute, Nagoya 464-8681, Japan. 2001 - 2008

References

Roles of achaete-scute homologue 1 in DKK1 and E-cadherin repression and neuroendocrine differentiation in lung cancer. Osada, H., Tomida, S., Yatabe, Y., Tatematsu, Y., Takeuchi, T., Murakami, H., Kondo, Y., Sekido, Y., Takahashi, T. Cancer Res. (2008) [Pubmed]
ASH1 gene is a specific therapeutic target for lung cancers with neuroendocrine features. Osada, H., Tatematsu, Y., Yatabe, Y., Horio, Y., Takahashi, T. Cancer Res. (2005) [Pubmed]
Reduced expression of class II histone deacetylase genes is associated with poor prognosis in lung cancer patients. Osada, H., Tatematsu, Y., Saito, H., Yatabe, Y., Mitsudomi, T., Takahashi, T. Int. J. Cancer (2004) [Pubmed]
Frequent and histological type-specific inactivation of 14-3-3sigma in human lung cancers. Osada, H., Tatematsu, Y., Yatabe, Y., Nakagawa, T., Konishi, H., Harano, T., Tezel, E., Takada, M., Takahashi, T. Oncogene (2002) [Pubmed]
Heterogeneous transforming growth factor (TGF)-beta unresponsiveness and loss of TGF-beta receptor type II expression caused by histone deacetylation in lung cancer cell lines. Osada, H., Tatematsu, Y., Masuda, A., Saito, T., Sugiyama, M., Yanagisawa, K., Takahashi, T. Cancer Res. (2001) [Pubmed]