Hosea F. S. Huang
Department of Surgery
Section of Urology
UMD-New Jersey Medical School
Newark
USA
Name/email consistency: high
- Stable expression of constitutively-activated STAT3 in benign prostatic epithelial cells changes their phenotype to that resembling malignant cells. Huang, H.F., Murphy, T.F., Shu, P., Barton, A.B., Barton, B.E. Mol. Cancer (2005)
- Preservation of spermatogenesis in spinal cord injured rats with exogenous testosterone. Relationship with serum testosterone levels and cellular localization of cAMP responsive element modulator. Huang, H.F., Wang, S., Molina, C.A., Ottenweller, J.E. J. Androl. (2004)
- Effects of exogenous testosterone on testicular function during the chronic phase of spinal cord injury: dose effects on spermatogenesis and Sertoli cell and sperm function. Huang, H.F., Li, M.T., Wang, S., Barton, B., Anesetti, R., Jetko, J.A. J. Spinal. Cord. Med (2004)
- Spinal cord contusion impairs sperm motility in the rat without disrupting spermatogenesis. Huang, H.F., Li, M.T., Wang, S., Wang, G., Ottenweller, J.E. J. Androl. (2003)
- Alteration of cyclic adenosine 3',5'-monophosphate signaling in rat testicular cells after spinal cord injury. Huang, H.F., Li, M.T., Wang, S., Pogach, L.M., Ottenweller, J.E. J. Spinal. Cord. Med (2003)
- Androgen and retinoic acid interaction in LNCaP cells, effects on cell proliferation and expression of retinoic acid receptors and epidermal growth factor receptor. Li, M.T., Richter, F., Chang, C., Irwin, R.J., Huang, H. BMC. Cancer (2002)
- The effects of testicular denervation on spermatogenesis in the Sprague-Dawley rat. Chow, S.H., Giglio, W., Anesetti, R., Ottenweller, J.E., Pogach, L.M., Huang, H.F. Neuroendocrinology (2000)
- Effects of spinal cord injury on spermatogenesis and the expression of messenger ribonucleic acid for Sertoli cell proteins in rat Sertoli cell-enriched testes. Huang, H.F., Li, M.T., Anesetti, R., Giglio, W., Ottenweller, J.E., Pogach, L.M. Biol. Reprod. (1999)
- The detrimental effects of spinal cord injury on spermatogenesis in the rat is partially reversed by testosterone, but enhanced by follicle-stimulating hormone. Huang, H.F., Li, M.T., Giglio, W., Anesetti, R., Ottenweller, J.E., Pogach, L.M. Endocrinology (1999)