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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)
 

James T. B. Crawley

Department of Haematology

Imperial College London

London

UK

[email]@imperial.ac.uk

Name/email consistency: high

 
 
 
 
 
 
 

Affiliations

  • Department of Haematology, Imperial College London, London, UK. 2007 - 2009
  • Department of Hematology, Imperial College London, 5th Floor Commonwealth Building, Hammersmith Hospital Campus, UK. 2008
  • Haematology Department, Division of Investigative Sciences, Faculty of Medicine, Imperial College London, United Kingdom. 2005

References

  1. Circulating ADAMTS-13-von Willebrand factor complexes: an enzyme on demand. Crawley, J.T., DE Groot, R., Luken, B.M. J. Thromb. Haemost. (2009) [Pubmed]
  2. Evidence that high von Willebrand factor and low ADAMTS-13 levels independently increase the risk of a non-fatal heart attack. Crawley, J.T., Lane, D.A., Woodward, M., Rumley, A., Lowe, G.D. J. Thromb. Haemost. (2008) [Pubmed]
  3. The haemostatic role of tissue factor pathway inhibitor. Crawley, J.T., Lane, D.A. Arterioscler. Thromb. Vasc. Biol. (2008) [Pubmed]
  4. Cytoprotective effect of activated protein C: specificity of PAR-1 signaling. Crawley, J.T., Efthymiou, M. J. Thromb. Haemost. (2008) [Pubmed]
  5. Multiple roles of the endothelial cell protein C receptor. Crawley, J.T. J. Thromb. Haemost. (2007) [Pubmed]
  6. Proteolytic inactivation of ADAMTS13 by thrombin and plasmin. Crawley, J.T., Lam, J.K., Rance, J.B., Mollica, L.R., O'Donnell, J.S., Lane, D.A. Blood (2005) [Pubmed]
 
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