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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)
 

Jacob Nattermann

Department of Internal Medicine I

University of Bonn

Germany

[email]@ukb.uni-bonn.de

Name/email consistency: high

 
 
 
 
 
 
 

Affiliation

  • Department of Internal Medicine I, University of Bonn, Germany. 2004 - 2008

References

  1. The transforming growth factor-beta high-producer genotype is associated with response to hepatitis C virus-specific therapy in HIV-positive patients with acute hepatitis C. Nattermann, J., Vogel, M., Nischalke, H.D., Danta, M., Ahlenstiel, G., Michalk, M., Sauerbruch, T., Rockstroh, J.K., Spengler, U. AIDS (2008) [Pubmed]
  2. Host genetic factors and treatment of hepatitis C. Nattermann, J., Leifeld, L., Spengler, U. Curr. Mol. Pharmacol (2008) [Pubmed]
  3. Effect of the interleukin-6 C174G gene polymorphism on treatment of acute and chronic hepatitis C in human immunodeficiency virus coinfected patients. Nattermann, J., Vogel, M., Berg, T., Danta, M., Axel, B., Mayr, C., Bruno, R., Tural, C., Klausen, G., Clotet, B., Lutz, T., Grünhage, F., Rausch, M., Nischalke, H.D., Schewe, K., Bienek, B., Haerter, G., Sauerbruch, T., Rockstroh, J.K., Spengler, U. Hepatology (2007) [Pubmed]
  4. Hepatitis C virus E2 and CD81 interaction may be associated with altered trafficking of dendritic cells in chronic hepatitis C. Nattermann, J., Zimmermann, H., Iwan, A., von Lilienfeld-Toal, M., Leifeld, L., Nischalke, H.D., Langhans, B., Sauerbruch, T., Spengler, U. Hepatology (2006) [Pubmed]
  5. Surface expression and cytolytic function of natural killer cell receptors is altered in chronic hepatitis C. Nattermann, J., Feldmann, G., Ahlenstiel, G., Langhans, B., Sauerbruch, T., Spengler, U. Gut (2006) [Pubmed]
  6. The tandem-repeat polymorphism of the DC-SIGNR gene in HCV infection. Nattermann, J., Ahlenstiel, G., Berg, T., Feldmann, G., Nischalke, H.D., Müller, T., Rockstroh, J., Woitas, R., Sauerbruch, T., Spengler, U. J. Viral Hepat. (2006) [Pubmed]
  7. The HLA-A2 restricted T cell epitope HCV core 35-44 stabilizes HLA-E expression and inhibits cytolysis mediated by natural killer cells. Nattermann, J., Nischalke, H.D., Hofmeister, V., Ahlenstiel, G., Zimmermann, H., Leifeld, L., Weiss, E.H., Sauerbruch, T., Spengler, U. Am. J. Pathol. (2005) [Pubmed]
  8. HIV-1 infection leads to increased HLA-E expression resulting in impaired function of natural killer cells. Nattermann, J., Nischalke, H.D., Hofmeister, V., Kupfer, B., Ahlenstiel, G., Feldmann, G., Rockstroh, J., Weiss, E.H., Sauerbruch, T., Spengler, U. Antivir. Ther. (Lond.) (2005) [Pubmed]
  9. Serum antibodies against the hepatitis C virus E2 protein mediate antibody-dependent cellular cytotoxicity (ADCC). Nattermann, J., Schneiders, A.M., Leifeld, L., Langhans, B., Schulz, M., Inchauspé, G., Matz, B., Brackmann, H.H., Houghton, M., Sauerbruch, T., Spengler, U. J. Hepatol. (2005) [Pubmed]
  10. Binding of HCV E2 to CD81 induces RANTES secretion and internalization of CC chemokine receptor 5. Nattermann, J., Nischalke, H.D., Feldmann, G., Ahlenstiel, G., Sauerbruch, T., Spengler, U. J. Viral Hepat. (2004) [Pubmed]
 
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