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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)
 

Jane A. Leopold

Whitaker Cardiovascular Institute and Evans Department of Medicine

Boston University School of Medicine

Boston

Massachusetts 02118

USA

[email]@bmc.org

Name/email consistency: high

 
 
 
 
 
 
 

Affiliation

  • Whitaker Cardiovascular Institute and Evans Department of Medicine, Boston University School of Medicine, Boston, Massachusetts 02118, USA. 2000 - 2003

References

  1. Glucose-6-phosphate dehydrogenase overexpression decreases endothelial cell oxidant stress and increases bioavailable nitric oxide. Leopold, J.A., Zhang, Y.Y., Scribner, A.W., Stanton, R.C., Loscalzo, J. Arterioscler. Thromb. Vasc. Biol. (2003) [Pubmed]
  2. Glucose-6-phosphate dehydrogenase modulates vascular endothelial growth factor-mediated angiogenesis. Leopold, J.A., Walker, J., Scribner, A.W., Voetsch, B., Zhang, Y.Y., Loscalzo, A.J., Stanton, R.C., Loscalzo, J. J. Biol. Chem. (2003) [Pubmed]
  3. Glucose-6-phosphate dehydrogenase deficiency promotes endothelial oxidant stress and decreases endothelial nitric oxide bioavailability. Leopold, J.A., Cap, A., Scribner, A.W., Stanton, R.C., Loscalzo, J. FASEB J. (2001) [Pubmed]
  4. Cyclic strain modulates resistance to oxidant stress by increasing G6PDH expression in smooth muscle cells. Leopold, J.A., Loscalzo, J. Am. J. Physiol. Heart Circ. Physiol. (2000) [Pubmed]
 
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