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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)
 

Jan M. Schwab

Institute of Brain Research

University of Tuebingen

Medical School

Tuebingen

Germany

[email]@med.uni-tuebingen.de

Name/email consistency: high

 
 
 
 
 
 
 

Affiliations

  • Institute of Brain Research, University of Tuebingen, Medical School, Tuebingen, Germany. 2000 - 2004
  • Department of Neuropathology, Neuroimmunology, Medical School, Institute of Brain Research, Germany. 2003

References

  1. Lesional RhoA+ cell numbers are suppressed by anti-inflammatory, cyclooxygenase-inhibiting treatment following subacute spinal cord injury. Schwab, J.M., Conrad, S., Elbert, T., Trautmann, K., Meyermann, R., Schluesener, H.J. Glia (2004) [Pubmed]
  2. COX-3: just another COX or the solitary elusive target of paracetamol?. Schwab, J.M., Schluesener, H.J., Laufer, S. Lancet (2003) [Pubmed]
  3. COX-3 the enzyme and the concept: steps towards highly specialized pathways and precision therapeutics?. Schwab, J.M., Schluesener, H.J., Meyermann, R., Serhan, C.N. Prostaglandins Leukot. Essent. Fatty Acids (2003) [Pubmed]
  4. COX-3--a virtual pain target in humans?. Schwab, J.M., Beiter, T., Linder, J.U., Laufer, S., Schulz, J.E., Meyermann, R., Schluesener, H.J. FASEB J. (2003) [Pubmed]
  5. Persistent accumulation of cyclooxygenase-1-expressing microglial cells and macrophages and transient upregulation by endothelium in human brain injury. Schwab, J.M., Beschorner, R., Meyermann, R., Gözalan, F., Schluesener, H.J. J. Neurosurg. (2002) [Pubmed]
  6. IL-16 is differentially expressed in the developing human fetal brain by microglial cells in zones of neuropoesis. Schwab, J.M., Schluesener, H.J., Seid, K., Meyermann, R. Int. J. Dev. Neurosci. (2001) [Pubmed]
  7. Human focal cerebral infarctions induce differential lesional interleukin-16 (IL-16) expression confined to infiltrating granulocytes, CD8+ T-lymphocytes and activated microglia/macrophages. Schwab, J.M., Nguyen, T.D., Meyermann, R., Schluesener, H.J. J. Neuroimmunol. (2001) [Pubmed]
  8. Differential cellular accumulation of connective tissue growth factor defines a subset of reactive astrocytes, invading fibroblasts, and endothelial cells following central nervous system injury in rats and humans. Schwab, J.M., Beschorner, R., Nguyen, T.D., Meyermann, R., Schluesener, H.J. J. Neurotrauma (2001) [Pubmed]
  9. Functional recovery after spinal cord injury: basic science meets clinic. Schwab, J.M., Leppert, C.A., Kaps, K.H., Monnier, P.P. Trends Neurosci. (2001) [Pubmed]
  10. Traumatic brain injury induces prolonged accumulation of cyclooxygenase-1 expressing microglia/brain macrophages in rats. Schwab, J.M., Seid, K., Schluesener, H.J. J. Neurotrauma (2001) [Pubmed]
  11. AIF-1 expression defines a proliferating and alert microglial/macrophage phenotype following spinal cord injury in rats. Schwab, J.M., Frei, E., Klusman, I., Schnell, L., Schwab, M.E., Schluesener, H.J. J. Neuroimmunol. (2001) [Pubmed]
  12. Connective tissue growth factor is expressed by a subset of reactive astrocytes in human cerebral infarction. Schwab, J.M., Postler, E., Nguyen, T.D., Mittelbronn, M., Meyermann, R., Schluesener, H.J. Neuropathol. Appl. Neurobiol. (2000) [Pubmed]
  13. Persistent accumulation of cyclooxygenase-1 (COX-1) expressing microglia/macrophages and upregulation by endothelium following spinal cord injury. Schwab, J.M., Brechtel, K., Nguyen, T.D., Schluesener, H.J. J. Neuroimmunol. (2000) [Pubmed]
 
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