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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)

John A. McGrath

St John's Institute of Dermatology

King's College London (Guy's Campus)

London SE1 9RT



Name/email consistency: high



  • St John's Institute of Dermatology, King's College London (Guy's Campus), London SE1 9RT, UK. 2010 - 2012
  • Genetic Skin Disease Group, St John's Institute of Dermatology, Division of Genetics and Molecular Medicine, The Guy's, UK. 2008
  • Department of Cell and Molecular Pathology, St John's Institute of Dermatology, The Guy's, King's College and St Thomas' Hospitals' Medical School, UK. 1999 - 2005


  1. Germline Mutation in EXPH5 Implicates the Rab27B Effector Protein Slac2-b in Inherited Skin Fragility. McGrath, J.A., Stone, K.L., Begum, R., Simpson, M.A., Dopping-Hepenstal, P.J., Liu, L., McMillan, J.R., South, A.P., Pourreyron, C., McLean, W.H., Martinez, A.E., Mellerio, J.E., Parsons, M. Am. J. Hum. Genet. (2012) [Pubmed]
  2. Profilaggrin, dry skin, and atopic dermatitis risk: size matters. McGrath, J.A. J. Invest. Dermatol. (2012) [Pubmed]
  3. Lethal acantholytic epidermolysis bullosa. McGrath, J.A., Bolling, M.C., Jonkman, M.F. Dermatol. Clin (2010) [Pubmed]
  4. Ectodermal dysplasia-skin fragility syndrome. McGrath, J.A., Mellerio, J.E. Dermatol. Clin (2010) [Pubmed]
  5. Filaggrin and the great epidermal barrier grief. McGrath, J.A. Australas. J. Dermatol. (2008) [Pubmed]
  6. Second cite. McGrath, J.A. Clin. Exp. Dermatol. (2006) [Pubmed]
  7. Inherited disorders of desmosomes. McGrath, J.A. Australas. J. Dermatol. (2005) [Pubmed]
  8. Translational benefits from research on rare genodermatoses. McGrath, J.A. Australas. J. Dermatol. (2004) [Pubmed]
  9. Recent advances in the molecular basis of inherited skin diseases. McGrath, J.A., Eady, R.A. Adv. Genet. (2001) [Pubmed]
  10. Keratinocyte adhesion and the missing link: from Dowling-Meara to Hay-Wells. St John's Hospital Dermatological Society Annual Oration 2000. McGrath, J.A. Clin. Exp. Dermatol. (2001) [Pubmed]
  11. Moderation of phenotypic severity in dystrophic and junctional forms of epidermolysis bullosa through in-frame skipping of exons containing non-sense or frameshift mutations. McGrath, J.A., Ashton, G.H., Mellerio, J.E., Salas-Alanis, J.C., Swensson, O., McMillan, J.R., Eady, R.A. J. Invest. Dermatol. (1999) [Pubmed]
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