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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)
 

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Joseph T. Coyle

*To whom correspondence should be addressed; tel: 617-855-2101

fax: 617-855-2705

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[email]@hms.harvard.edu

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Affiliations

  • *To whom correspondence should be addressed; tel: 617-855-2101, fax: 617-855-2705, e-mail:. 2012
  • Department of Psychiatry, Harvard Medical School, The Laboratory of Molecular Psychiatry and Neuroscience, McLean Hospital, USA. 2012
  • Department of Psychiatry, Harvard Medical School, McLean Hospital, Belmont, USA. 2000 - 2010
  • McLean Hospital, Belmont, Massachusetts 02478, USA. 2003

References

  1. NMDA Receptor and Schizophrenia: A Brief History. Coyle, J.T. Schizophr. Bull (2012) [Pubmed]
  2. Glutamatergic synaptic dysregulation in schizophrenia: therapeutic implications. Coyle, J.T., Basu, A., Benneyworth, M., Balu, D., Konopaske, G. Handb. Exp. Pharmacol (2012) [Pubmed]
  3. Beyond the dopamine receptor: novel therapeutic targets for treating schizophrenia. Coyle, J.T., Balu, D., Benneyworth, M., Basu, A., Roseman, A. Dialogues. Clin. Neurosci (2010) [Pubmed]
  4. MicroRNAs suggest a new mechanism for altered brain gene expression in schizophrenia. Coyle, J.T. Proc. Natl. Acad. Sci. U.S.A. (2009) [Pubmed]
  5. Beyond in vitro data: a review of in vivo evidence regarding the allosteric potentiating effect of galantamine on nicotinic acetylcholine receptors in Alzheimer's neuropathology. Coyle, J.T., Geerts, H., Sorra, K., Amatniek, J. J. Alzheimers Dis. (2007) [Pubmed]
  6. What can a clock mutation in mice tell us about bipolar disorder?. Coyle, J.T. Proc. Natl. Acad. Sci. U.S.A. (2007) [Pubmed]
  7. Glial metabolites of tryptophan and excitotoxicity: coming unglued. Coyle, J.T. Exp. Neurol. (2006) [Pubmed]
  8. Glutamate and schizophrenia: beyond the dopamine hypothesis. Coyle, J.T. Cell. Mol. Neurobiol. (2006) [Pubmed]
  9. A brief overview of N-acetylaspartate and N-acetylaspartylglutamate. Coyle, J.T. Adv. Exp. Med. Biol. (2006) [Pubmed]
  10. Substance use disorders and Schizophrenia: a question of shared glutamatergic mechanisms. Coyle, J.T. Neurotox. Res (2006) [Pubmed]
  11. The NMDA receptor glycine modulatory site: a therapeutic target for improving cognition and reducing negative symptoms in schizophrenia. Coyle, J.T., Tsai, G. Psychopharmacology (Berl.) (2004) [Pubmed]
  12. The GABA-glutamate connection in schizophrenia: which is the proximate cause?. Coyle, J.T. Biochem. Pharmacol. (2004) [Pubmed]
  13. Finding the intracellular signaling pathways affected by mood disorder treatments. Coyle, J.T., Duman, R.S. Neuron (2003) [Pubmed]
  14. Converging evidence of NMDA receptor hypofunction in the pathophysiology of schizophrenia. Coyle, J.T., Tsai, G., Goff, D. Ann. N. Y. Acad. Sci. (2003) [Pubmed]
  15. Ionotropic glutamate receptors as therapeutic targets in schizophrenia. Coyle, J.T., Tsai, G., Goff, D.C. Curr. Drug. Targets. CNS. Neurol. Disord (2002) [Pubmed]
  16. Mind glue: implications of glial cell biology for psychiatry. Coyle, J.T., Schwarcz, R. Arch. Gen. Psychiatry (2000) [Pubmed]
 
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