Hideaki Kaneto
Department of Metabolic Medicine
Osaka University Graduate School of Medicine
Suita
Osaka 565-0871
Japan
Name/email consistency: high
- Combination of MafA, PDX-1 and NeuroD is a useful tool to efficiently induce insulin-producing surrogate beta-cells. Kaneto, H., Matsuoka, T.A., Katakami, N., Matsuhisa, M. Curr. Med. Chem. (2009)
- Role of MafA in pancreatic beta-cells. Kaneto, H., Matsuoka, T.A., Kawashima, S., Yamamoto, K., Kato, K., Miyatsuka, T., Katakami, N., Matsuhisa, M. Adv. Drug Deliv. Rev. (2009)
- PDX-1 functions as a master factor in the pancreas. Kaneto, H., Matsuoka, T.A., Miyatsuka, T., Kawamori, D., Katakami, N., Yamasaki, Y., Matsuhisa, M. Front. Biosci. (2008)
- Oxidative stress and the JNK pathway are involved in the development of type 1 and type 2 diabetes. Kaneto, H., Matsuoka, T.A., Katakami, N., Kawamori, D., Miyatsuka, T., Yoshiuchi, K., Yasuda, T., Sakamoto, K., Yamasaki, Y., Matsuhisa, M. Curr. Mol. Med. (2007)
- Crucial role of PDX-1 in pancreas development, beta-cell differentiation, and induction of surrogate beta-cells. Kaneto, H., Miyatsuka, T., Shiraiwa, T., Yamamoto, K., Kato, K., Fujitani, Y., Matsuoka, T.A. Curr. Med. Chem. (2007)
- Role of PDX-1 and MafA as a potential therapeutic target for diabetes. Kaneto, H., Miyatsuka, T., Fujitani, Y., Noguchi, H., Song, K.H., Yoon, K.H., Matsuoka, T.A. Diabetes Res. Clin. Pract. (2007)
- Involvement of oxidative stress in the pathogenesis of diabetes. Kaneto, H., Katakami, N., Kawamori, D., Miyatsuka, T., Sakamoto, K., Matsuoka, T.A., Matsuhisa, M., Yamasaki, Y. Antioxid. Redox Signal. (2007)
- A crucial role of MafA as a novel therapeutic target for diabetes. Kaneto, H., Matsuoka, T.A., Nakatani, Y., Miyatsuka, T., Matsuhisa, M., Hori, M., Yamasaki, Y. J. Biol. Chem. (2005)
- Oxidative stress, ER stress, and the JNK pathway in type 2 diabetes. Kaneto, H., Matsuoka, T.A., Nakatani, Y., Kawamori, D., Miyatsuka, T., Matsuhisa, M., Yamasaki, Y. J. Mol. Med. (2005)
- PDX-1/VP16 fusion protein, together with NeuroD or Ngn3, markedly induces insulin gene transcription and ameliorates glucose tolerance. Kaneto, H., Nakatani, Y., Miyatsuka, T., Matsuoka, T.A., Matsuhisa, M., Hori, M., Yamasaki, Y. Diabetes (2005)
- Role of oxidative stress, endoplasmic reticulum stress, and c-Jun N-terminal kinase in pancreatic beta-cell dysfunction and insulin resistance. Kaneto, H., Nakatani, Y., Kawamori, D., Miyatsuka, T., Matsuoka, T.A., Matsuhisa, M., Yamasaki, Y. Int. J. Biochem. Cell Biol. (2005)
- The JNK pathway as a therapeutic target for diabetes. Kaneto, H. Expert Opin. Ther. Targets (2005)
- Oxidative stress and pancreatic beta-cell dysfunction. Kaneto, H., Kawamori, D., Matsuoka, T.A., Kajimoto, Y., Yamasaki, Y. Am. J. Ther (2005)
- Possible novel therapy for diabetes with cell-permeable JNK-inhibitory peptide. Kaneto, H., Nakatani, Y., Miyatsuka, T., Kawamori, D., Matsuoka, T.A., Matsuhisa, M., Kajimoto, Y., Ichijo, H., Yamasaki, Y., Hori, M. Nat. Med. (2004)
- Oxidative stress and the JNK pathway as a potential therapeutic target for diabetes. Kaneto, H., Kawamori, D., Nakatani, Y., Gorogawa, S., Matsuoka, T.A. Drug News Perspect. (2004)
- Involvement of oxidative stress and the JNK pathway in glucose toxicity. Kaneto, H., Nakatani, Y., Kawamori, D., Miyatsuka, T., Matsuoka, T.A. Review. Diabetic. Studies. :. Rds (2004)
