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Karl M. Stuhlmeier

Ludwig Boltzmann Institute for Rheumatology and Balneology

Kurbadstrasse 10

1100 Vienna

Austria

[email]@excite.com

Name/email consistency: high

 
 
 
 
 
 
 

Affiliations

  • Ludwig Boltzmann Institute for Rheumatology and Balneology, Kurbadstrasse 10, 1100 Vienna, Austria. 2001 - 2008
  • Center for Immunobiology, Beth Israel Deaconess Medical Center, Harvard Medical School, Boston, USA. 2000

References

  1. Hyaluronan production in synoviocytes as a consequence of viral infections: HAS1 activation by Epstein-Barr virus and synthetic double- and single-stranded viral RNA analogs. Stuhlmeier, K.M. J. Biol. Chem. (2008) [Pubmed]
  2. Apis mellifera venom and melittin block neither NF-kappa B-p50-DNA interactions nor the activation of NF-kappa B, instead they activate the transcription of proinflammatory genes and the release of reactive oxygen intermediates. Stuhlmeier, K.M. J. Immunol. (2007) [Pubmed]
  3. Prostaglandin E2: a potent activator of hyaluronan synthase 1 in type-B-synoviocytes. Stuhlmeier, K.M. Biochim. Biophys. Acta (2007) [Pubmed]
  4. The anti-rheumatic gold salt aurothiomalate suppresses interleukin-1beta-induced hyaluronan accumulation by blocking HAS1 transcription and by acting as a COX-2 transcriptional repressor. Stuhlmeier, K.M. J. Biol. Chem. (2007) [Pubmed]
  5. Quinacrine but not chloroquine inhibits PMA induced upregulation of matrix metalloproteinases in leukocytes: quinacrine acts at the transcriptional level through a PLA2-independent mechanism. Stuhlmeier, K.M., Pollaschek, C. J. Rheumatol. (2006) [Pubmed]
  6. Aspects of the biology of hyaluronan, a largely neglected but extremely versatile molecule. Stuhlmeier, K.M. Wien. Med. Wochenschr (2006) [Pubmed]
  7. Effects of leflunomide on hyaluronan synthases (HAS): NF-kappa B-independent suppression of IL-1-induced HAS1 transcription by leflunomide. Stuhlmeier, K.M. J. Immunol. (2005) [Pubmed]
  8. Adenovirus-mediated gene transfer of mutated IkappaB kinase and IkappaBalpha reveal NF-kappaB-dependent as well as NF-kappaB-independent pathways of HAS1 activation. Stuhlmeier, K.M., Pollaschek, C. J. Biol. Chem. (2005) [Pubmed]
  9. Glucocorticoids inhibit induced and non-induced mRNA accumulation of genes encoding hyaluronan synthases (HAS): hydrocortisone inhibits HAS1 activation by blocking the p38 mitogen-activated protein kinase signalling pathway. Stuhlmeier, K.M., Pollaschek, C. Rheumatology. (Oxford) (2004) [Pubmed]
  10. Differential effect of transforming growth factor beta (TGF-beta) on the genes encoding hyaluronan synthases and utilization of the p38 MAPK pathway in TGF-beta-induced hyaluronan synthase 1 activation. Stuhlmeier, K.M., Pollaschek, C. J. Biol. Chem. (2004) [Pubmed]
  11. Antioxidant protein 2 prevents methemoglobin formation in erythrocyte hemolysates. Stuhlmeier, K.M., Kao, J.J., Wallbrandt, P., Lindberg, M., Hammarström, B., Broell, H., Paigen, B. Eur. J. Biochem. (2003) [Pubmed]
  12. Mepacrine inhibits matrix metalloproteinases-1 (MMP-1) and MMP-9 activation in human fibroblast-like synoviocytes. Stuhlmeier, K.M. J. Rheumatol. (2003) [Pubmed]
  13. Effects of quinacrine on endothelial cell morphology and transcription factor-DNA interactions. Stuhlmeier, K.M. Biochim. Biophys. Acta (2001) [Pubmed]
  14. Activation and regulation of Hsp32 and Hsp70. Stuhlmeier, K.M. Eur. J. Biochem. (2000) [Pubmed]
 
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