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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)
 

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Kenneth I. Ataga

Comprehensive Sickle Cell Program Division of Hematology/Oncology Department of Biochemistry and Biophysics Department of Biostatistics Department of Pediatrics

University of North Carolina

Chapel Hill

NC 27599-7305

USA

[email]@med.unc.edu

Name/email consistency: high

 
 
 
 
 
 
 

Affiliations

  • Comprehensive Sickle Cell Program Division of Hematology/Oncology Department of Biochemistry and Biophysics Department of Biostatistics Department of Pediatrics, University of North Carolina, Chapel Hill, NC 27599-7305, USA. 2011
  • Comprehensive Sickle Cell Program, University of North Carolina, Chapel Hill, USA. 2010
  • Division of Hematology/Oncology, University of North Carolina at Chapel Hill, Room 3149, Physicians Office Building, USA. 2004 - 2009
  • Division of Hematology/Oncology, Department of Medicine, UNC Comprehensive Sickle Cell Program & General Clinical Research Center, University of North Carolina, USA. 2000 - 2006

References

  1. Association of soluble fms-like tyrosine kinase-1 with pulmonary hypertension and haemolysis in sickle cell disease. Ataga, K.I., Brittain, J.E., Jones, S.K., May, R., Delaney, J., Strayhorn, D., Desai, P., Redding-Lallinger, R., Key, N.S., Orringer, E.P. Br. J. Haematol. (2011) [Pubmed]
  2. Urinary albumin excretion is associated with pulmonary hypertension in sickle cell disease: potential role of soluble fms-like tyrosine kinase-1. Ataga, K.I., Brittain, J.E., Moore, D., Jones, S.K., Hulkower, B., Strayhorn, D., Adam, S., Redding-Lallinger, R., Nachman, P., Orringer, E.P. Eur. J. Haematol. (2010) [Pubmed]
  3. Senicapoc (ICA-17043): a potential therapy for the prevention and treatment of hemolysis-associated complications in sickle cell anemia. Ataga, K.I., Stocker, J. Expert. Opin. Investig. Drugs (2009) [Pubmed]
  4. Novel therapies in sickle cell disease. Ataga, K.I. Hematology. Am. Soc. Hematol. Educ. Program (2009) [Pubmed]
  5. Efficacy and safety of the Gardos channel blocker, senicapoc (ICA-17043), in patients with sickle cell anemia. Ataga, K.I., Smith, W.R., De Castro, L.M., Swerdlow, P., Saunthararajah, Y., Castro, O., Vichinsky, E., Kutlar, A., Orringer, E.P., Rigdon, G.C., Stocker, J.W. Blood (2008) [Pubmed]
  6. Coagulation activation and inflammation in sickle cell disease-associated pulmonary hypertension. Ataga, K.I., Moore, C.G., Hillery, C.A., Jones, S., Whinna, H.C., Strayhorn, D., Sohier, C., Hinderliter, A., Parise, L.V., Orringer, E.P. Haematologica (2008) [Pubmed]
  7. Beta-thalassaemia and sickle cell anaemia as paradigms of hypercoagulability. Ataga, K.I., Cappellini, M.D., Rachmilewitz, E.A. Br. J. Haematol. (2007) [Pubmed]
  8. Hypercoagulability in sickle cell disease: new approaches to an old problem. Ataga, K.I., Key, N.S. Hematology. Am. Soc. Hematol. Educ. Program (2007) [Pubmed]
  9. Pulmonary hypertension in patients with sickle cell disease: a longitudinal study. Ataga, K.I., Moore, C.G., Jones, S., Olajide, O., Strayhorn, D., Hinderliter, A., Orringer, E.P. Br. J. Haematol. (2006) [Pubmed]
  10. Dose-escalation study of ICA-17043 in patients with sickle cell disease. Ataga, K.I., Orringer, E.P., Styles, L., Vichinsky, E.P., Swerdlow, P., Davis, G.A., Desimone, P.A., Stocker, J.W. Pharmacotherapy (2006) [Pubmed]
  11. Pulmonary hypertension in sickle cell disease. Ataga, K.I., Sood, N., De Gent, G., Kelly, E., Henderson, A.G., Jones, S., Strayhorn, D., Lail, A., Lieff, S., Orringer, E.P. Am. J. Med. (2004) [Pubmed]
  12. Hypercoagulability in sickle cell disease: a curious paradox. Ataga, K.I., Orringer, E.P. Am. J. Med. (2003) [Pubmed]
  13. Renal abnormalities in sickle cell disease. Ataga, K.I., Orringer, E.P. Am. J. Hematol. (2000) [Pubmed]
 
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