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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)

Michael J. Katovich

M. J. Katovich: Department of Pharmadocynamics

University of Florida

PO Box 100487

SW 1600 Archer Road



Name/email consistency: high



  • M. J. Katovich: Department of Pharmadocynamics, University of Florida, PO Box 100487, SW 1600 Archer Road, USA. 2008 - 2011
  • Department of Pharmacodynamics, College of Pharmacy, University of Florida, Gainesville 32610, USA. 2000 - 2005


  1. Lentivirus-mediated overexpression of angiotensin-(1-7) attenuated ischaemia-induced cardiac pathophysiology. Qi, Y., Shenoy, V., Wong, F., Li, H., Afzal, A., Mocco, J., Sumners, C., Raizada, M.K., Katovich, M.J. Exp. Physiol. (2011) [Pubmed]
  2. Angiotensin-(1-7) as an antihypertensive, antifibrotic target. Katovich, M.J., Grobe, J.L., Raizada, M.K. Curr. Hypertens. Rep. (2008) [Pubmed]
  3. Angiotensin-converting enzyme 2 as a novel target for gene therapy for hypertension. Katovich, M.J., Grobe, J.L., Huentelman, M., Raizada, M.K. Exp. Physiol. (2005) [Pubmed]
  4. Gene therapy attenuates the elevated blood pressure and glucose intolerance in an insulin-resistant model of hypertension. Katovich, M.J., Reaves, P.Y., Francis, S.C., Pachori, A.S., Wang, H.W., Raizada, M.K. J. Hypertens. (2001) [Pubmed]
  5. Effects of inhibition of the renin-angiotensin system on the cardiovascular actions of insulin. Katovich, M.J., Pachori, A. Diabetes. Obes. Metab (2000) [Pubmed]
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