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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)
 

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Keshore R. Bidasee

Dept. of Pharmacology and Experimental Neuroscience

Univ. of Nebraska Medical Center

DRC 3047

Omaha

USA

[email]@unmc.edu

Name/email consistency: high

 
 
 
 
 
 
 

Affiliations

  • Dept. of Pharmacology and Experimental Neuroscience, Univ. of Nebraska Medical Center, DRC 3047, Omaha, USA. 2003 - 2008
  • Department of Pharmacology and Toxicology, Indiana University School of Medicine, Indianapolis, USA. 2003

References

  1. Exercise training initiated after the onset of diabetes preserves myocardial function: effects on expression of beta-adrenoceptors. Bidasee, K.R., Zheng, H., Shao, C.H., Parbhu, S.K., Rozanski, G.J., Patel, K.P. J. Appl. Physiol. (2008) [Pubmed]
  2. Diabetes increases formation of advanced glycation end products on Sarco(endo)plasmic reticulum Ca2+-ATPase. Bidasee, K.R., Zhang, Y., Shao, C.H., Wang, M., Patel, K.P., Dincer, U.D., Besch, H.R. Diabetes (2004) [Pubmed]
  3. Diketopyridylryanodine has three concentration-dependent effects on the cardiac calcium-release channel/ryanodine receptor. Bidasee, K.R., Xu, L., Meissner, G., Besch, H.R. J. Biol. Chem. (2003) [Pubmed]
  4. Streptozotocin-induced diabetes increases disulfide bond formation on cardiac ryanodine receptor (RyR2). Bidasee, K.R., Nallani, K., Besch, H.R., Dincer, U.D. J. Pharmacol. Exp. Ther. (2003) [Pubmed]
  5. Chronic diabetes increases advanced glycation end products on cardiac ryanodine receptors/calcium-release channels. Bidasee, K.R., Nallani, K., Yu, Y., Cocklin, R.R., Zhang, Y., Wang, M., Dincer, U.D., Besch, H.R. Diabetes (2003) [Pubmed]
  6. Chronic diabetes alters function and expression of ryanodine receptor calcium-release channels in rat hearts. Bidasee, K.R., Nallani, K., Henry, B., Dincer, U.D., Besch, H.R. Mol. Cell. Biochem. (2003) [Pubmed]
 
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