Kirsten L. White
Gilead Sciences
Foster City
California
94404
USA
Name/email consistency: high
- The K65R reverse transcriptase mutation in HIV-1 reverses the excision phenotype of zidovudine resistance mutations. White, K.L., Chen, J.M., Feng, J.Y., Margot, N.A., Ly, J.K., Ray, A.S., Macarthur, H.L., McDermott, M.J., Swaminathan, S., Miller, M.D. Antivir. Ther. (Lond.) (2006)
- A combination of decreased NRTI incorporation and decreased excision determines the resistance profile of HIV-1 K65R RT. White, K.L., Margot, N.A., Ly, J.K., Chen, J.M., Ray, A.S., Pavelko, M., Wang, R., McDermott, M., Swaminathan, S., Miller, M.D. AIDS (2005)
- Molecular mechanisms of tenofovir resistance conferred by human immunodeficiency virus type 1 reverse transcriptase containing a diserine insertion after residue 69 and multiple thymidine analog-associated mutations. White, K.L., Chen, J.M., Margot, N.A., Wrin, T., Petropoulos, C.J., Naeger, L.K., Swaminathan, S., Miller, M.D. Antimicrob. Agents Chemother. (2004)