Lihua Bao
Section of Nephrology
The University of Chicago
5841 S. Maryland Avenue
MC5100
USA
Name/email consistency: high
- Complement factor H deficiency accelerates development of lupus nephritis. Bao, L., Haas, M., Quigg, R.J. J. Am. Soc. Nephrol. (2011)
- Focal and segmental glomerulosclerosis induced in mice lacking decay-accelerating factor in T cells. Bao, L., Haas, M., Pippin, J., Wang, Y., Miwa, T., Chang, A., Minto, A.W., Petkova, M., Qiao, G., Song, W.C., Alpers, C.E., Zhang, J., Shankland, S.J., Quigg, R.J. J. Clin. Invest. (2009)
- Mesangial cell complement receptor 1-related protein y limits complement-dependent neutrophil accumulation in immune complex glomerulonephritis. Bao, L., Wang, Y., Chen, P., Sarav, M., Haas, M., Minto, A.W., Petkova, M., Quigg, R.J. Immunology (2009)
- Decay-accelerating factor but not CD59 limits experimental immune-complex glomerulonephritis. Bao, L., Haas, M., Minto, A.W., Quigg, R.J. Lab. Invest. (2007)
- Complement in lupus nephritis: the good, the bad, and the unknown. Bao, L., Quigg, R.J. Semin. Nephrol. (2007)
- Signaling through up-regulated C3a receptor is key to the development of experimental lupus nephritis. Bao, L., Osawe, I., Haas, M., Quigg, R.J. J. Immunol. (2005)
- C5a promotes development of experimental lupus nephritis which can be blocked with a specific receptor antagonist. Bao, L., Osawe, I., Puri, T., Lambris, J.D., Haas, M., Quigg, R.J. Eur. J. Immunol. (2005)
