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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)

Loren G. Fong

Department of Medicine

University of California

Los Angeles

CA 90095



Name/email consistency: high



  • Department of Medicine, University of California, Los Angeles, CA 90095, USA. 2004 - 2009
  • Department of Medicine/Division of Cardiology, David Geffen School of Medicine, University of California, Los Angeles, USA. 2006


  1. Activating the synthesis of progerin, the mutant prelamin A in Hutchinson-Gilford progeria syndrome, with antisense oligonucleotides. Fong, L.G., Vickers, T.A., Farber, E.A., Choi, C., Yun, U.J., Hu, Y., Yang, S.H., Coffinier, C., Lee, R., Yin, L., Davies, B.S., Andres, D.A., Spielmann, H.P., Bennett, C.F., Young, S.G. Hum. Mol. Genet. (2009) [Pubmed]
  2. A protein farnesyltransferase inhibitor ameliorates disease in a mouse model of progeria. Fong, L.G., Frost, D., Meta, M., Qiao, X., Yang, S.H., Coffinier, C., Young, S.G. Science (2006) [Pubmed]
  3. Prelamin A and lamin A appear to be dispensable in the nuclear lamina. Fong, L.G., Ng, J.K., Lammerding, J., Vickers, T.A., Meta, M., Coté, N., Gavino, B., Qiao, X., Chang, S.Y., Young, S.R., Yang, S.H., Stewart, C.L., Lee, R.T., Bennett, C.F., Bergo, M.O., Young, S.G. J. Clin. Invest. (2006) [Pubmed]
  4. Heterozygosity for Lmna deficiency eliminates the progeria-like phenotypes in Zmpste24-deficient mice. Fong, L.G., Ng, J.K., Meta, M., Coté, N., Yang, S.H., Stewart, C.L., Sullivan, T., Burghardt, A., Majumdar, S., Reue, K., Bergo, M.O., Young, S.G. Proc. Natl. Acad. Sci. U.S.A. (2004) [Pubmed]
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