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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)
 

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Linda Dondero Hazlett

Department of Anatomy and Cell Biology

Wayne State University School of Medicine

Detroit

MI 48201

USA

[email]@med.wayne.edu

Name/email consistency: high

 
 
 
 
 
 
 

Affiliation

  • Department of Anatomy and Cell Biology, Wayne State University School of Medicine, Detroit, MI 48201, USA. 1999 - 2010

References

  1. Reviews for immune privilege in the year 2010: immune privilege and infection. Hazlett, L.D., Hendricks, R.L. Ocul. Immunol. Inflamm. (2010) [Pubmed]
  2. IL-33 shifts macrophage polarization, promoting resistance against Pseudomonas aeruginosa keratitis. Hazlett, L.D., McClellan, S.A., Barrett, R.P., Huang, X., Zhang, Y., Wu, M., van Rooijen, N., Szliter, E. Invest. Ophthalmol. Vis. Sci. (2010) [Pubmed]
  3. Bacterial infections of the cornea (Pseudomonas aeruginosa). Hazlett, L.D. Chem. Immunol. Allergy (2007) [Pubmed]
  4. NKT cells are critical to initiate an inflammatory response after Pseudomonas aeruginosa ocular infection in susceptible mice. Hazlett, L.D., Li, Q., Liu, J., McClellan, S., Du, W., Barrett, R.P. J. Immunol. (2007) [Pubmed]
  5. ST2 is essential for Th2 responsiveness and resistance to pseudomonas aeruginosa keratitis. Huang, X., Du, W., Barrett, R.P., Hazlett, L.D. Invest. Ophthalmol. Vis. Sci. (2007) [Pubmed]
  6. Spantide I decreases type I cytokines, enhances IL-10, and reduces corneal perforation in susceptible mice after Pseudomonas aeruginosa infection. Hazlett, L.D., McClellan, S.A., Barrett, R.P., Liu, J., Zhang, Y., Lighvani, S. Invest. Ophthalmol. Vis. Sci. (2007) [Pubmed]
  7. The role of nitric oxide in resistance to P. aeruginosa ocular infection. Hazlett, L.D., McClellan, S., Goshgarian, C., Huang, X., Thakur, A., Barrett, R. Ocul. Immunol. Inflamm. (2005) [Pubmed]
  8. Inflammatory response to Pseudomonas aeruginosa keratitis. Hazlett, L.D. Ocul. Surf (2005) [Pubmed]
  9. Corneal response to Pseudomonas aeruginosa infection. Hazlett, L.D. Prog. Retin. Eye. Res (2004) [Pubmed]
  10. Further studies on the role of IL-12 in Pseudomonas aeruginosa corneal infection. Hazlett, L.D., Huang, X., McClellan, S.A., Barrett, R.P. Eye. (Lond) (2003) [Pubmed]
  11. The role of Langerhans cells in Pseudomonas aeruginosa infection. Hazlett, L.D., McClellan, S.A., Rudner, X.L., Barrett, R.P. Invest. Ophthalmol. Vis. Sci. (2002) [Pubmed]
  12. Role of IL-12 and IFN-gamma in Pseudomonas aeruginosa corneal infection. Hazlett, L.D., Rudner, X.L., McClellan, S.A., Barrett, R.P., Lighvani, S. Invest. Ophthalmol. Vis. Sci. (2002) [Pubmed]
  13. Pathogenic mechanisms of P. aeruginosa keratitis: a review of the role of T cells, Langerhans cells, PMN, and cytokines. Hazlett, L.D. DNA Cell Biol. (2002) [Pubmed]
  14. B7/CD28 costimulation is critical in susceptibility to Pseudomonas aeruginosa corneal infection: a comparative study using monoclonal antibody blockade and CD28-deficient mice. Hazlett, L.D., McClellan, S., Barrett, R., Rudner, X. J. Immunol. (2001) [Pubmed]
  15. Increased severity of Pseudomonas aeruginosa corneal infection in strains of mice designated as Th1 versus Th2 responsive. Hazlett, L.D., McClellan, S., Kwon, B., Barrett, R. Invest. Ophthalmol. Vis. Sci. (2000) [Pubmed]
  16. Complement defects in aged mice compromise phagocytosis of Pseudomonas aeruginosa. Hazlett, L.D., Masinick-McClellan, S.A., Barrett, R.P. Curr. Eye Res. (1999) [Pubmed]
 
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