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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)
 

Sally A. Litherland

Department of Pathology

Immunology

and Laboratory Medicine

University of Florida

USA

[email]@pathology.ufl.edu

Name/email consistency: high

 
 
 
 
 
 
 

Affiliation

  • Department of Pathology, Immunology, and Laboratory Medicine, University of Florida, USA. 2004 - 2005

References

  1. Signal transduction activator of transcription 5 (STAT5) dysfunction in autoimmune monocytes and macrophages. Litherland, S.A., Xie, T.X., Grebe, K.M., Davoodi-Semiromi, A., Elf, J., Belkin, N.S., Moldawer, L.L., Clare-Salzler, M.J. J. Autoimmun. (2005) [Pubmed]
  2. Nonobese diabetic mouse congenic analysis reveals chromosome 11 locus contributing to diabetes susceptibility, macrophage STAT5 dysfunction, and granulocyte-macrophage colony-stimulating factor overproduction. Litherland, S.A., Grebe, K.M., Belkin, N.S., Paek, E., Elf, J., Atkinson, M., Morel, L., Clare-Salzler, M.J., McDuffie, M. J. Immunol. (2005) [Pubmed]
  3. IL10 resistant PGS2 expression in at-risk/Type 1 diabetic human monocytes. Litherland, S.A., Xie, T.X., Grebe, K.M., Li, Y., Moldawer, L.L., Clare-Salzler, M.J. J. Autoimmun. (2004) [Pubmed]
 
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