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Elizabeth A. Woodcock

Molecular Cardiology Laboratory

Baker IDI Heart and Diabetes Institute

PO Box 6492

St Kilda Road Central

Australia

[email]@baker.edu.au

Name/email consistency: high

 
 
 
 
 
 
 

Affiliations

  • Molecular Cardiology Laboratory, Baker IDI Heart and Diabetes Institute, PO Box 6492, St Kilda Road Central, Australia. 2009
  • Cellular Biochemistry Laboratory, Baker Heart Research Institute, Melbourne, Victoria, Australia. 1997 - 2007

References

  1. Phosphoinositide signalling and cardiac arrhythmias. Woodcock, E.A., Kistler, P.M., Ju, Y.K. Cardiovasc. Res. (2009) [Pubmed]
  2. Roles of alpha1A- and alpha1B-adrenoceptors in heart: insights from studies of genetically modified mice. Woodcock, E.A. Clin. Exp. Pharmacol. Physiol. (2007) [Pubmed]
  3. Ins(1,4,5)P3 receptors and inositol phosphates in the heart-evolutionary artefacts or active signal transducers?. Woodcock, E.A., Matkovich, S.J. Pharmacol. Ther. (2005) [Pubmed]
  4. Phospholipase Cdelta(1) does not mediate Ca(2+) responses in neonatal rat cardiomyocytes. Woodcock, E.A., Mitchell, C.J., Biden, T.J. FEBS Lett. (2003) [Pubmed]
  5. Inositol polyphosphate 1-phosphatase is a novel antihypertrophic factor. Woodcock, E.A., Wang, B.H., Arthur, J.F., Lennard, A., Matkovich, S.J., Du, X.J., Brown, J.H., Hannan, R.D. J. Biol. Chem. (2002) [Pubmed]
  6. Reperfusion-induced Ins(1,4,5)P(3) generation and arrhythmogenesis require activation of the Na(+)/Ca(2+) exchanger. Woodcock, E.A., Arthur, J.F., Harrison, S.N., Gao, X.M., Du, X.J. J. Mol. Cell. Cardiol. (2001) [Pubmed]
  7. Inositol 1,4,5-trisphosphate and reperfusion arrhythmias. Woodcock, E.A., Arthur, J.F., Matkovich, S.J. Clin. Exp. Pharmacol. Physiol. (2000) [Pubmed]
  8. Ins(1,4,5)P3 and cardiac dysfunction. Woodcock, E.A., Matkovich, S.J., Binah, O. Cardiovasc. Res. (1998) [Pubmed]
  9. Inositol phosphates and inositol phospholipids: how big is the iceberg?. Woodcock, E.A. Mol. Cell. Endocrinol. (1997) [Pubmed]
 
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