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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)

Elizabeth A. Woodcock

Molecular Cardiology Laboratory

Baker IDI Heart and Diabetes Institute

PO Box 6492

St Kilda Road Central



Name/email consistency: high



  • Molecular Cardiology Laboratory, Baker IDI Heart and Diabetes Institute, PO Box 6492, St Kilda Road Central, Australia. 2009
  • Cellular Biochemistry Laboratory, Baker Heart Research Institute, Melbourne, Victoria, Australia. 1997 - 2007


  1. Phosphoinositide signalling and cardiac arrhythmias. Woodcock, E.A., Kistler, P.M., Ju, Y.K. Cardiovasc. Res. (2009) [Pubmed]
  2. Roles of alpha1A- and alpha1B-adrenoceptors in heart: insights from studies of genetically modified mice. Woodcock, E.A. Clin. Exp. Pharmacol. Physiol. (2007) [Pubmed]
  3. Ins(1,4,5)P3 receptors and inositol phosphates in the heart-evolutionary artefacts or active signal transducers?. Woodcock, E.A., Matkovich, S.J. Pharmacol. Ther. (2005) [Pubmed]
  4. Phospholipase Cdelta(1) does not mediate Ca(2+) responses in neonatal rat cardiomyocytes. Woodcock, E.A., Mitchell, C.J., Biden, T.J. FEBS Lett. (2003) [Pubmed]
  5. Inositol polyphosphate 1-phosphatase is a novel antihypertrophic factor. Woodcock, E.A., Wang, B.H., Arthur, J.F., Lennard, A., Matkovich, S.J., Du, X.J., Brown, J.H., Hannan, R.D. J. Biol. Chem. (2002) [Pubmed]
  6. Reperfusion-induced Ins(1,4,5)P(3) generation and arrhythmogenesis require activation of the Na(+)/Ca(2+) exchanger. Woodcock, E.A., Arthur, J.F., Harrison, S.N., Gao, X.M., Du, X.J. J. Mol. Cell. Cardiol. (2001) [Pubmed]
  7. Inositol 1,4,5-trisphosphate and reperfusion arrhythmias. Woodcock, E.A., Arthur, J.F., Matkovich, S.J. Clin. Exp. Pharmacol. Physiol. (2000) [Pubmed]
  8. Ins(1,4,5)P3 and cardiac dysfunction. Woodcock, E.A., Matkovich, S.J., Binah, O. Cardiovasc. Res. (1998) [Pubmed]
  9. Inositol phosphates and inositol phospholipids: how big is the iceberg?. Woodcock, E.A. Mol. Cell. Endocrinol. (1997) [Pubmed]
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