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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)
 

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R. Lodi

Dipartimento di Medicina Clinica e Biotecnologia Applicata D. Campanacci

Università di Bologna

Bologna

Italy

[email]@med.unibo.it

Name/email consistency: high

 
 
 
 
 
 
 

Affiliations

  • Dipartimento di Medicina Clinica e Biotecnologia Applicata D. Campanacci, Università di Bologna, Bologna, Italy. 2000 - 2003
  • Medical Research Council, Biochemical and Clinical Magnetic Resonance Unit, Department of Biochemistry, University of Oxford. 1999 - 2001
  • Department of Biochemistry, University of Oxford, United Kingdom. 2001

References

  1. Gliomatosis cerebri: clinical, neurochemical and neuroradiological response to temozolomide administration. Lodi, R., Setola, E., Tonon, C., Ambrosetto, P., Franceschi, E., Crinò, L., Barbiroli, B., Cortelli, P. Magn. Reson. Imaging (2003) [Pubmed]
  2. Phosphorus MR spectroscopy shows a tissue specific in vivo distribution of biochemical expression of the G3460A mutation in Leber's hereditary optic neuropathy. Lodi, R., Carelli, V., Cortelli, P., Iotti, S., Valentino, M.L., Barboni, P., Pallotti, F., Montagna, P., Barbiroli, B. J. Neurol. Neurosurg. Psychiatr. (2002) [Pubmed]
  3. Mitochondrial dysfunction in Friedreich's ataxia: from pathogenesis to treatment perspectives. Lodi, R., Rajagopalan, B., Bradley, J.L., Taylor, D.J., Crilley, J.G., Hart, P.E., Blamire, A.M., Manners, D., Styles, P., Schapira, A.H., Cooper, J.M. Free Radic. Res. (2002) [Pubmed]
  4. Deficient energy metabolism is associated with low free magnesium in the brains of patients with migraine and cluster headache. Lodi, R., Iotti, S., Cortelli, P., Pierangeli, G., Cevoli, S., Clementi, V., Soriani, S., Montagna, P., Barbiroli, B. Brain Res. Bull. (2001) [Pubmed]
  5. Mitochondrial dysfunction in friedreich's ataxia. Lodi, R., Taylor, D.J., Schapira, A.H. Biol. Signals. Recept (2001) [Pubmed]
  6. Antioxidant treatment improves in vivo cardiac and skeletal muscle bioenergetics in patients with Friedreich's ataxia. Lodi, R., Hart, P.E., Rajagopalan, B., Taylor, D.J., Crilley, J.G., Bradley, J.L., Blamire, A.M., Manners, D., Styles, P., Schapira, A.H., Cooper, J.M. Ann. Neurol. (2001) [Pubmed]
  7. Cardiac energetics are abnormal in Friedreich ataxia patients in the absence of cardiac dysfunction and hypertrophy: an in vivo 31P magnetic resonance spectroscopy study. Lodi, R., Rajagopalan, B., Blamire, A.M., Cooper, J.M., Davies, C.H., Bradley, J.L., Styles, P., Schapira, A.H. Cardiovasc. Res. (2001) [Pubmed]
  8. 'Secondary' 4216/ND1 and 13708/ND5 Leber's hereditary optic neuropathy mitochondrial DNA mutations do not further impair in vivo mitochondrial oxidative metabolism when associated with the 11778/ND4 mitochondrial DNA mutation. Lodi, R., Montagna, P., Cortelli, P., Iotti, S., Cevoli, S., Carelli, V., Barbiroli, B. Brain (2000) [Pubmed]
  9. Deficit of in vivo mitochondrial ATP production in patients with Friedreich ataxia. Lodi, R., Cooper, J.M., Bradley, J.L., Manners, D., Styles, P., Taylor, D.J., Schapira, A.H. Proc. Natl. Acad. Sci. U.S.A. (1999) [Pubmed]
 
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