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Lorraine B. Ware

Division of Allergy

Pulmonary and Critical Care Medicine

Department of Medicine

Vanderbilt University School of Medicine

USA

[email]@vanderbilt.edu

Name/email consistency: high

 
 
 
 
 
 
 

Affiliations

  • Division of Allergy, Pulmonary and Critical Care Medicine, Department of Medicine, Vanderbilt University School of Medicine, USA. 2002 - 2012
  • T1218 Medical Center North, Nashville, TN 37232-2650. 2012
  • Vanderbilt Univ. School of Medicine, Nashville, TN 37232-2650, USA. 2003

References

  1. Comparison of chest radiograph scoring to lung weight as a quantitative index of pulmonary edema in organ donors. Ware, L.B., Neyrinck, A., O'Neal, H.R., Lee, J.W., Landeck, M., Johnson, E., Calfee, C.S., Matthay, M.A. Clin. Transplant (2012) [Pubmed]
  2. The severity of shock is associated with impaired rates of net alveolar fluid clearance in clinical acute lung injury. Zeyed, Y.F., Bastarache, J.A., Matthay, M.A., Ware, L.B. Am. J. Physiol. Lung Cell Mol. Physiol. (2012) [Pubmed]
  3. Prognostic and pathogenetic value of combining clinical and biochemical indices in patients with acute lung injury. Ware, L.B., Koyama, T., Billheimer, D.D., Wu, W., Bernard, G.R., Thompson, B.T., Brower, R.G., Standiford, T.J., Martin, T.R., Matthay, M.A. Chest (2010) [Pubmed]
  4. Determining the aetiology of pulmonary oedema by the oedema fluid-to-plasma protein ratio. Ware, L.B., Fremont, R.D., Bastarache, J.A., Calfee, C.S., Matthay, M.A. Eur. Respir. J. (2010) [Pubmed]
  5. Clinical year in review I: Interstitial lung disease, pulmonary vascular disease, pulmonary infections, and cardiopulmonary exercise testing and pulmonary rehabilitation. Ware, L.B. Proc. Am. Thorac. Soc (2009) [Pubmed]
  6. Clinical year in review II: Sepsis, mechanical ventilation, occupational and environmental lung disease, and sleep. Ware, L.B., Eisner, M.D., Flaherty, K.R. Proc. Am. Thorac. Soc (2009) [Pubmed]
  7. Pathogenetic and prognostic significance of altered coagulation and fibrinolysis in acute lung injury/acute respiratory distress syndrome. Ware, L.B., Matthay, M.A., Parsons, P.E., Thompson, B.T., Januzzi, J.L., Eisner, M.D. Crit. Care Med. (2007) [Pubmed]
  8. Bench to bedside: targeting coagulation and fibrinolysis in acute lung injury. Ware, L.B., Camerer, E., Welty-Wolf, K., Schultz, M.J., Matthay, M.A. Am. J. Physiol. Lung Cell Mol. Physiol. (2006) [Pubmed]
  9. Pathophysiology of acute lung injury and the acute respiratory distress syndrome. Ware, L.B. Semin. Respir. Crit. Care. Med (2006) [Pubmed]
  10. VEGF levels in the alveolar compartment do not distinguish between ARDS and hydrostatic pulmonary oedema. Ware, L.B., Kaner, R.J., Crystal, R.G., Schane, R., Trivedi, N.N., McAuley, D., Matthay, M.A. Eur. Respir. J. (2005) [Pubmed]
  11. High prevalence of pulmonary arterial thrombi in donor lungs rejected for transplantation. Ware, L.B., Fang, X., Wang, Y., Babcock, W.D., Jones, K., Matthay, M.A. J. Heart Lung Transplant. (2005) [Pubmed]
  12. Coagulation and fibrinolysis in human acute lung injury--new therapeutic targets?. Ware, L.B., Bastarache, J.A., Wang, L. Keio. J. Med (2005) [Pubmed]
  13. Significance of von Willebrand factor in septic and nonseptic patients with acute lung injury. Ware, L.B., Eisner, M.D., Thompson, B.T., Parsons, P.E., Matthay, M.A. Am. J. Respir. Crit. Care Med. (2004) [Pubmed]
  14. Protein C and thrombomodulin in human acute lung injury. Ware, L.B., Fang, X., Matthay, M.A. Am. J. Physiol. Lung Cell Mol. Physiol. (2003) [Pubmed]
  15. Assessment of lungs rejected for transplantation and implications for donor selection. Ware, L.B., Wang, Y., Fang, X., Warnock, M., Sakuma, T., Hall, T.S., Matthay, M. Lancet (2002) [Pubmed]
  16. Selected contribution: mechanisms that may stimulate the resolution of alveolar edema in the transplanted human lung. Ware, L.B., Fang, X., Wang, Y., Sakuma, T., Hall, T.S., Matthay, M.A. J. Appl. Physiol. (2002) [Pubmed]
 
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