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Luis B. Agellon

Department of Biochemistry and Canadian Institutes of Health Research Group in Molecular and Cell Biology of Lipids

University of Alberta

Edmonton

Alberta T6G 2S2

Canada

[email]@ualberta.ca

Name/email consistency: high

 
 
 
 
 
 
 

Affiliations

  • Department of Biochemistry and Canadian Institutes of Health Research Group in Molecular and Cell Biology of Lipids, University of Alberta, Edmonton, Alberta T6G 2S2, Canada. 2002 - 2006
  • Department of Biochemistry, University of Alberta, Edmonton, Canada. 1997 - 2000

References

  1. Adaptations to the loss of intestinal fatty acid binding protein in mice. Agellon, L.B., Li, L., Luong, L., Uwiera, R.R. Mol. Cell. Biochem. (2006) [Pubmed]
  2. Dietary cholesterol fails to stimulate the human cholesterol 7alpha-hydroxylase gene (CYP7A1) in transgenic mice. Agellon, L.B., Drover, V.A., Cheema, S.K., Gbaguidi, G.F., Walsh, A. J. Biol. Chem. (2002) [Pubmed]
  3. Intracellular lipid binding proteins of the small intestine. Agellon, L.B., Toth, M.J., Thomson, A.B. Mol. Cell. Biochem. (2002) [Pubmed]
  4. Intracellular transport of bile acids. Agellon, L.B., Torchia, E.C. Biochim. Biophys. Acta (2000) [Pubmed]
  5. The unique acyl chain specificity of biliary phosphatidylcholines in mice is independent of their biosynthetic origin in the liver. Agellon, L.B., Walkey, C.J., Vance, D.E., Kuipers, F., Verkade, H.J. Hepatology (1999) [Pubmed]
  6. Partial transfection of liver with a synthetic cholesterol 7 alpha-hydroxylase transgene is sufficient to stimulate the reduction of cholesterol in the plasma of hypercholesterolemic mice. Agellon, L.B. Biochem. Cell Biol. (1997) [Pubmed]
 
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