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Daniela Macconi

Mario Negri Institute for Pharmacological Research

Department of Biomedical Engineering

Laboratory of Renal Biophysics

Via Gavazzeni

Italy

[email]@marionegri.it

Name/email consistency: high

 
 
 
 
 
 
 

Affiliations

  • Mario Negri Institute for Pharmacological Research, Department of Biomedical Engineering, Laboratory of Renal Biophysics, Via Gavazzeni, Italy. 2009
  • Mario Negri Institute for Pharmacological Research, Via Gavazzeni 11, 24125 Bergamo, Italy. 2006

References

  1. Podocyte repopulation contributes to regression of glomerular injury induced by ACE inhibition. Macconi, D., Sangalli, F., Bonomelli, M., Conti, S., Condorelli, L., Gagliardini, E., Remuzzi, G., Remuzzi, A. Am. J. Pathol. (2009) [Pubmed]
  2. Pathophysiologic implications of reduced podocyte number in a rat model of progressive glomerular injury. Macconi, D., Bonomelli, M., Benigni, A., Plati, T., Sangalli, F., Longaretti, L., Conti, S., Kawachi, H., Hill, P., Remuzzi, G., Remuzzi, A. Am. J. Pathol. (2006) [Pubmed]
  3. Permselective dysfunction of podocyte-podocyte contact upon angiotensin II unravels the molecular target for renoprotective intervention. Macconi, D., Abbate, M., Morigi, M., Angioletti, S., Mister, M., Buelli, S., Bonomelli, M., Mundel, P., Endlich, K., Remuzzi, A., Remuzzi, G. Am. J. Pathol. (2006) [Pubmed]
 
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