Daniela Macconi
Mario Negri Institute for Pharmacological Research
Department of Biomedical Engineering
Laboratory of Renal Biophysics
Via Gavazzeni
Italy
Name/email consistency: high
- Podocyte repopulation contributes to regression of glomerular injury induced by ACE inhibition. Macconi, D., Sangalli, F., Bonomelli, M., Conti, S., Condorelli, L., Gagliardini, E., Remuzzi, G., Remuzzi, A. Am. J. Pathol. (2009)
- Pathophysiologic implications of reduced podocyte number in a rat model of progressive glomerular injury. Macconi, D., Bonomelli, M., Benigni, A., Plati, T., Sangalli, F., Longaretti, L., Conti, S., Kawachi, H., Hill, P., Remuzzi, G., Remuzzi, A. Am. J. Pathol. (2006)
- Permselective dysfunction of podocyte-podocyte contact upon angiotensin II unravels the molecular target for renoprotective intervention. Macconi, D., Abbate, M., Morigi, M., Angioletti, S., Mister, M., Buelli, S., Bonomelli, M., Mundel, P., Endlich, K., Remuzzi, A., Remuzzi, G. Am. J. Pathol. (2006)