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Michael Madeja

Institute of Physiology

University of Münster

Münster

Germany

[email]@ghst.de

Name/email consistency: high

 
 
 
 
 
 
 

Affiliation

  • Institute of Physiology, University of Münster, Münster, Germany. 2001 - 2010

References

  1. Overlapping binding sites of structurally different antiarrhythmics flecainide and propafenone in the subunit interface of potassium channel Kv2.1. Madeja, M., Steffen, W., Mesic, I., Garic, B., Zhorov, B.S. J. Biol. Chem. (2010) [Pubmed]
  2. Molecular site of action of the antiarrhythmic drug propafenone at the voltage-operated potassium channel Kv2.1. Madeja, M., Leicher, T., Friederich, P., Punke, M.A., Haverkamp, W., Musshoff, U., Breithardt, G., Speckmann, E.J. Mol. Pharmacol. (2003) [Pubmed]
  3. Reduction of voltage-operated potassium currents by levetiracetam: a novel antiepileptic mechanism of action?. Madeja, M., Margineanu, D.G., Gorji, A., Siep, E., Boerrigter, P., Klitgaard, H., Speckmann, E.J. Neuropharmacology (2003) [Pubmed]
  4. Reduction of voltage-operated sodium currents by the anticonvulsant drug sulthiame. Madeja, M., Wolf, C., Speckmann, E.J. Brain Res. (2001) [Pubmed]
 
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