Maria G. Frid
Developmental Lung Biology Research Laboratory
Department of Pediatrics
University of Colorado Health Sciences Center
Denver 80262
USA
Name/email consistency: high
- Hypoxia-induced pulmonary vascular remodeling requires recruitment of circulating mesenchymal precursors of a monocyte/macrophage lineage. Frid, M.G., Brunetti, J.A., Burke, D.L., Carpenter, T.C., Davie, N.J., Reeves, J.T., Roedersheimer, M.T., van Rooijen, N., Stenmark, K.R. Am. J. Pathol. (2006)
- Circulating mononuclear cells with a dual, macrophage-fibroblast phenotype contribute robustly to hypoxia-induced pulmonary adventitial remodeling. Frid, M.G., Brunetti, J.A., Burke, D.L., Carpenter, T.C., Davie, N.J., Stenmark, K.R. Chest (2005)
- Yin and Yang of an endothelial cell: from normal to the extreme in growth, secretion, and transdifferentiation capabilities. Frid, M.G., Aldashev, A.A., Crossno, J.T., Jorgensen, J.M., Kale, V.A., Stenmark, K.R. Paediatr. Respir. Rev (2004)
- Mature vascular endothelium can give rise to smooth muscle cells via endothelial-mesenchymal transdifferentiation: in vitro analysis. Frid, M.G., Kale, V.A., Stenmark, K.R. Circ. Res. (2002)
- Subendothelial cells from normal bovine arteries exhibit autonomous growth and constitutively activated intracellular signaling. Frid, M.G., Aldashev, A.A., Nemenoff, R.A., Higashito, R., Westcott, J.Y., Stenmark, K.R. Arterioscler. Thromb. Vasc. Biol. (1999)