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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)
 

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Lee J. Martin

Department of Pathology

Division of Neuropathology

Johns Hopkins University School of Medicine

558 Ross Building

USA

[email]@jhmi.edu

Name/email consistency: high

 
 
 
 
 
 
 

Affiliations

  • Department of Pathology, Division of Neuropathology, Johns Hopkins University School of Medicine, 558 Ross Building, USA. 2003 - 2012
  • Department of Pathology, 558 Ross Building, 720 Rutland Avenue, Baltimore, USA. 2011
  • Johns Hopkins University School of Medicine, Departments of Pathology and Neuroscience, Baltimore, MD 21205-2196, USA. 2010

References

  1. Inhibitory synaptic regulation of motoneurons: a new target of disease mechanisms in amyotrophic lateral sclerosis. Martin, L.J., Chang, Q. Mol. Neurobiol. (2012) [Pubmed]
  2. Generation and characterization of transgenic mice expressing mitochondrial targeted red fluorescent protein selectively in neurons: modeling mitochondriopathy in excitotoxicity and amyotrophic lateral sclerosis. Wang, Y., Pan, Y., Price, A., Martin, L.J. Mol. Neurodegener (2011) [Pubmed]
  3. An approach to experimental synaptic pathology using green fluorescent protein-transgenic mice and gene knockout mice to show mitochondrial permeability transition pore-driven excitotoxicity in interneurons and motoneurons. Martin, L.J. Toxicol. Pathol (2011) [Pubmed]
  4. The mitochondrial permeability transition pore regulates nitric oxide-mediated apoptosis of neurons induced by target deprivation. Martin, L.J., Adams, N.A., Pan, Y., Price, A., Wong, M. J. Neurosci. (2011) [Pubmed]
  5. The mitochondrial permeability transition pore: a molecular target for amyotrophic lateral sclerosis therapy. Martin, L.J. Biochim. Biophys. Acta (2010) [Pubmed]
  6. Olesoxime, a cholesterol-like neuroprotectant for the potential treatment of amyotrophic lateral sclerosis. Martin, L.J. IDrugs (2010) [Pubmed]
  7. Molecular regulation of DNA damage-induced apoptosis in neurons of cerebral cortex. Martin, L.J., Liu, Z., Pipino, J., Chestnut, B., Landek, M.A. Cereb. Cortex (2009) [Pubmed]
  8. The mitochondrial permeability transition pore in motor neurons: involvement in the pathobiology of ALS mice. Martin, L.J., Gertz, B., Pan, Y., Price, A.C., Molkentin, J.D., Chang, Q. Exp. Neurol. (2009) [Pubmed]
  9. DNA damage and repair: relevance to mechanisms of neurodegeneration. Martin, L.J. J. Neuropathol. Exp. Neurol. (2008) [Pubmed]
  10. Adult olfactory bulb neural precursor cell grafts provide temporary protection from motor neuron degeneration, improve motor function, and extend survival in amyotrophic lateral sclerosis mice. Martin, L.J., Liu, Z. J. Neuropathol. Exp. Neurol. (2007) [Pubmed]
  11. Transgenic mice with human mutant genes causing Parkinson's disease and amyotrophic lateral sclerosis provide common insight into mechanisms of motor neuron selective vulnerability to degeneration. Martin, L.J. Rev. Neurosci (2007) [Pubmed]
  12. Motor neuron degeneration in amyotrophic lateral sclerosis mutant superoxide dismutase-1 transgenic mice: mechanisms of mitochondriopathy and cell death. Martin, L.J., Liu, Z., Chen, K., Price, A.C., Pan, Y., Swaby, J.A., Golden, W.C. J. Comp. Neurol. (2007) [Pubmed]
  13. Parkinson's disease alpha-synuclein transgenic mice develop neuronal mitochondrial degeneration and cell death. Martin, L.J., Pan, Y., Price, A.C., Sterling, W., Copeland, N.G., Jenkins, N.A., Price, D.L., Lee, M.K. J. Neurosci. (2006) [Pubmed]
  14. Mitochondriopathy in Parkinson disease and amyotrophic lateral sclerosis. Martin, L.J. J. Neuropathol. Exp. Neurol. (2006) [Pubmed]
  15. Early events of target deprivation/axotomy-induced neuronal apoptosis in vivo: oxidative stress, DNA damage, p53 phosphorylation and subcellular redistribution of death proteins. Martin, L.J., Price, A.C., McClendon, K.B., Al-Abdulla, N.A., Subramaniam, J.R., Wong, P.C., Liu, Z. J. Neurochem. (2003) [Pubmed]
 
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