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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)
 

Mark A. Smith

Institute of Pathology

Case Western Reserve University

Cleveland

OH 44106

USA

[email]@po.cwru.edu

Name/email consistency: high

 
 
 
 
 
 
 

Affiliation

  • Institute of Pathology, Case Western Reserve University, Cleveland, OH 44106, USA. 1997 - 2003

References

  1. JKK1, an upstream activator of JNK/SAPK, is activated in Alzheimer's disease. Zhu, X., Ogawa, O., Wang, Y., Perry, G., Smith, M.A. J. Neurochem. (2003) [Pubmed]
  2. Amyloid-beta, tau alterations and mitochondrial dysfunction in Alzheimer disease: the chickens or the eggs?. Smith, M.A., Drew, K.L., Nunomura, A., Takeda, A., Hirai, K., Zhu, X., Atwood, C.S., Raina, A.K., Rottkamp, C.A., Sayre, L.M., Friedland, R.P., Perry, G. Neurochem. Int. (2002) [Pubmed]
  3. Causes and consequences of oxidative stress in Alzheimer's disease. Smith, M.A., Perry, G., Pryor, W.A. Free Radic. Biol. Med. (2002) [Pubmed]
  4. Amyloid-beta and tau serve antioxidant functions in the aging and Alzheimer brain. Smith, M.A., Casadesus, G., Joseph, J.A., Perry, G. Free Radic. Biol. Med. (2002) [Pubmed]
  5. Oxidative stress in Alzheimer's disease. Smith, M.A., Rottkamp, C.A., Nunomura, A., Raina, A.K., Perry, G. Biochim. Biophys. Acta (2000) [Pubmed]
  6. Metabolic, metallic, and mitotic sources of oxidative stress in Alzheimer disease. Smith, M.A., Nunomura, A., Zhu, X., Takeda, A., Perry, G. Antioxid. Redox Signal. (2000) [Pubmed]
  7. Diet and oxidative stress: a novel synthesis of epidemiological data on Alzheimer's disease. Smith, M.A., Petot, G.J., Perry, G. J. Alzheimers Dis. (1999) [Pubmed]
  8. Abnormal localization of iron regulatory protein in Alzheimer's disease. Smith, M.A., Wehr, K., Harris, P.L., Siedlak, S.L., Connor, J.R., Perry, G. Brain Res. (1998) [Pubmed]
  9. Dimethylargininase, a nitric oxide regulatory protein, in Alzheimer disease. Smith, M.A., Vasák, M., Knipp, M., Castellani, R.J., Perry, G. Free Radic. Biol. Med. (1998) [Pubmed]
  10. Iron accumulation in Alzheimer disease is a source of redox-generated free radicals. Smith, M.A., Harris, P.L., Sayre, L.M., Perry, G. Proc. Natl. Acad. Sci. U.S.A. (1997) [Pubmed]
 
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