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Andrea I. McClatchey

Massachusetts General Hospital Center for Cancer Research and Harvard Medical School Department of Pathology

Charlestown

MA, USA. Electronic address:

[email]@*.mgh.harvard.edu

Name/email consistency: high

 
 
 
 
 
 
 

Affiliations

  • Massachusetts General Hospital Center for Cancer Research and Harvard Medical School Department of Pathology, Charlestown, MA, USA. Electronic address:. 2000 - 2012
  • Center for Cancer Research, Massachusetts General Hospital, Charlestown, Massachusetts 02129, USA. 2007
  • Department of Biology and the Massachusetts Institute of Technology, Cambridge, Massachusetts 02139, USA. 1998
  • Massachusetts General Hospital Cancer Center, Charlestown 02129, USA. 1998

References

  1. Contact inhibition (of proliferation) redux. McClatchey, A.I., Yap, A.S. Curr. Opin. Cell Biol. (2012) [Pubmed]
  2. Neurofibromatosis. McClatchey, A.I. Annu. Rev. Pathol (2007) [Pubmed]
  3. Membrane organization and tumorigenesis--the NF2 tumor suppressor, Merlin. McClatchey, A.I., Giovannini, M. Genes Dev. (2005) [Pubmed]
  4. NF2 deficiency promotes tumorigenesis and metastasis by destabilizing adherens junctions. Lallemand, D., Curto, M., Saotome, I., Giovannini, M., McClatchey, A.I. Genes Dev. (2003) [Pubmed]
  5. Merlin and ERM proteins: unappreciated roles in cancer development?. McClatchey, A.I. Nat. Rev. Cancer (2003) [Pubmed]
  6. Mouse models of neurofibromatosis. McClatchey, A.I., Cichowski, K. Biochim. Biophys. Acta (2001) [Pubmed]
  7. Neurofibromatosis type II: mouse models reveal broad roles in tumorigenesis and metastasis. McClatchey, A.I. Mol. Med. Today (2000) [Pubmed]
  8. Mice heterozygous for a mutation at the Nf2 tumor suppressor locus develop a range of highly metastatic tumors. McClatchey, A.I., Saotome, I., Mercer, K., Crowley, D., Gusella, J.F., Bronson, R.T., Jacks, T. Genes Dev. (1998) [Pubmed]
  9. Tumor suppressor mutations in mice: the next generation. McClatchey, A.I., Jacks, T. Curr. Opin. Genet. Dev. (1998) [Pubmed]
 
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