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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)
 

David C. McMillan

Department of Cell and Molecular Pharmacology and Experimental Therapeutics

Medical University of South Carolina

Charleston 29425

USA

[email]@musc.edu

Name/email consistency: high

 
 
 
 
 
 
 

Affiliation

  • Department of Cell and Molecular Pharmacology and Experimental Therapeutics, Medical University of South Carolina, Charleston 29425, USA. 1999 - 2005

References

  1. Lipids versus proteins as major targets of pro-oxidant, direct-acting hemolytic agents. McMillan, D.C., Powell, C.L., Bowman, Z.S., Morrow, J.D., Jollow, D.J. Toxicol. Sci. (2005) [Pubmed]
  2. Role of oxidant stress in lawsone-induced hemolytic anemia. McMillan, D.C., Sarvate, S.D., Oatis, J.E., Jollow, D.J. Toxicol. Sci. (2004) [Pubmed]
  3. Favism: effect of divicine on rat erythrocyte sulfhydryl status, hexose monophosphate shunt activity, morphology, and membrane skeletal proteins. McMillan, D.C., Bolchoz, L.J., Jollow, D.J. Toxicol. Sci. (2001) [Pubmed]
  4. Favism: divicine hemotoxicity in the rat. McMillan, D.C., Jollow, D.J. Toxicol. Sci. (1999) [Pubmed]
 
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