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Michael Bitzer

Abteilung Innere Medizin I

Medizinische Universitätsklinik Tübingen

Germany

[email]@uni-tuebingen.de

Name/email consistency: high

 
 
 
 
 
 
 

Affiliations

  • Abteilung Innere Medizin I, Medizinische Universitätsklinik Tübingen, Germany. 1997 - 2003
  • Department of Internal Medicine I, University Clinic Tübingen, D-72076 Tübingen, Germany. 2002 - 2003

References

  1. Negative-strand RNA viral vectors: intravenous application of Sendai virus vectors for the systemic delivery of therapeutic genes. Bitzer, M., Ungerechts, G., Bossow, S., Graepler, F., Sedlmeier, R., Armeanu, S., Bernloehr, C., Spiegel, M., Gross, C.D., Gregor, M., Neubert, W.J., Lauer, U.M. Mol. Ther. (2003) [Pubmed]
  2. Sendai virus vectors as an emerging negative-strand RNA viral vector system. Bitzer, M., Armeanu, S., Lauer, U.M., Neubert, W.J. J. Gene. Med (2003) [Pubmed]
  3. A young woman with splenic infarction. Bitzer, M., Armeanu, S., Kröber, S.M., Horger, M.S., Erley, C.M. Lancet (2003) [Pubmed]
  4. Caspase-8 and Apaf-1-independent caspase-9 activation in Sendai virus-infected cells. Bitzer, M., Armeanu, S., Prinz, F., Ungerechts, G., Wybranietz, W., Spiegel, M., Bernlöhr, C., Cecconi, F., Gregor, M., Neubert, W.J., Schulze-Osthoff, K., Lauer, U.M. J. Biol. Chem. (2002) [Pubmed]
  5. Sendai virus infection induces apoptosis through activation of caspase-8 (FLICE) and caspase-3 (CPP32). Bitzer, M., Prinz, F., Bauer, M., Spiegel, M., Neubert, W.J., Gregor, M., Schulze-Osthoff, K., Lauer, U. J. Virol. (1999) [Pubmed]
  6. Sendai virus efficiently infects cells via the asialoglycoprotein receptor and requires the presence of cleaved F0 precursor proteins for this alternative route of cell entry. Bitzer, M., Lauer, U., Baumann, C., Spiegel, M., Gregor, M., Neubert, W.J. J. Virol. (1997) [Pubmed]
 
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