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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)
 

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Steven N. Mink

Health Sciences Centre

GF-221

820 Sherbrook St.

Winnipeg

Canada

[email]@cc.umanitoba.ca

Name/email consistency: high

 
 
 
 
 
 
 

Affiliations

  • Health Sciences Centre, GF-221, 820 Sherbrook St., Winnipeg, Canada. 2008 - 2011
  • Department of Medicine, University of Manitoba, Winnipeg, Manitoba, Canada. 1999 - 2009
  • Section of Respiratory, Department of Internal Medicine, University of Manitoba Health Sciences Centre, 700 William Avenue, Canada. 2004

References

  1. Ethyl gallate, a scavenger of hydrogen peroxide that inhibits lysozyme-induced hydrogen peroxide signaling in vitro, reverses hypotension in canine septic shock. Mink, S.N., Jacobs, H., Gotes, J., Kasian, K., Cheng, Z.Q. J. Appl. Physiol. (2011) [Pubmed]
  2. Lysozyme, a mediator of sepsis that intrinsically generates hydrogen peroxide to cause cardiovascular dysfunction. Mink, S.N., Jacobs, H., Cheng, Z.Q., Kasian, K., Santos-Martinez, L.E., Light, R.B. Am. J. Physiol. Heart Circ. Physiol. (2009) [Pubmed]
  3. Lysozyme, a mediator of sepsis that produces vasodilation by hydrogen peroxide signaling in an arterial preparation. Mink, S.N., Kasian, K., Santos Martinez, L.E., Jacobs, H., Bose, R., Cheng, Z.Q., Light, R.B. Am. J. Physiol. Heart Circ. Physiol. (2008) [Pubmed]
  4. N,N'-diacetylchitobiose, an inhibitor of lysozyme, reverses myocardial depression and lessens norepinephrine requirements in Escherichia coli sepsis in dogs. Mink, S.N., Kasian, K., Jacobs, H., Cheng, Z.Q., Light, R.B. Shock (2008) [Pubmed]
  5. Lysozyme, a mediator of sepsis, impairs the cardiac neural adrenergic response by nonendothelial release of NO and inhibitory G protein signaling. Mink, S.N., Cheng, Z.Q., Bose, R., Jacobs, H., Kasian, K., Roberts, D.E., Santos-Martinez, L.E., Light, R.B. Am. J. Physiol. Heart Circ. Physiol. (2007) [Pubmed]
  6. Lysozyme binding to endocardial endothelium mediates myocardial depression by the nitric oxide guanosine 3',5' monophosphate pathway in sepsis. Mink, S.N., Bose, R., Roberts, D.E., Jacobs, H., Duke, K., Bose, D., Cheng, Z.Q., Light, R.B. J. Mol. Cell. Cardiol. (2005) [Pubmed]
  7. N,N',N"-triacetylglucosamine, an inhibitor of lysozyme, prevents myocardial depression in Escherichia coli sepsis in dogs. Mink, S.N., Jacobs, H., Duke, K., Bose, D., Cheng, Z.Q., Light, R.B. Crit. Care Med. (2004) [Pubmed]
  8. Lung volume reduction surgery in canine model of predominantly upper lobe emphysema: advantages of new surgical system. Mink, S.N., Gonzalez, X., Duke, K., Bautista, E., Tan, L. Chest (2004) [Pubmed]
  9. Constant infusion of epinephrine, but not bolus treatment, improves haemodynamic recovery in anaphylactic shock in dogs. Mink, S.N., Simons, F.E., Simons, K.J., Becker, A.B., Duke, K. Clin. Exp. Allergy (2004) [Pubmed]
  10. Lysozyme: a mediator of myocardial depression and adrenergic dysfunction in septic shock in dogs. Mink, S.N., Jacobs, H., Bose, D., Duke, K., Cheng, Z.Q., Liu, G., Light, R.B. J. Mol. Cell. Cardiol. (2003) [Pubmed]
  11. Early but not delayed continuous arteriovenous hemofiltration improves cardiovascular function in sepsis in dogs. Mink, S.N., Li, X., Bose, D., Gu, M., Liu, G., Jacobs, H., Light, R.B. Intensive. Care. Med (1999) [Pubmed]
  12. Role of autacoids in cardiovascular colapse in anaphylactic shock in anaesthetized dogs. Mink, S., Becker, A., Sharma, S., Unruh, H., Duke, K., Kepron, W. Cardiovasc. Res. (1999) [Pubmed]
 
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